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- BookOlivier Walusinski.Contents:
Origins and family life
Education, medical studies, medical practice
Secretary, colleague, and friend of Jean-Martin Charcot
The assassination attempt
Glimpses of Gilles de la Tourette's personality : hospital life and the driout scandal
Gilles de la Tourette, chief physician for the 1900 World's Fair in Paris
A sad end : eclipse, twilight, and death
Doctoral thesis
Gilles de la Tourette syndrome
Vibratory medicine and therapeutic suspension techniques
L'hypnotisme et les états analogues (hypnotism and analogous states)
Clinical and therapeutic treatise on hysteria : 'hystérie normale'
Clinical and therapeutic treatise on hysteria : 'paroxysmic hysteria'
Soeur Jeanne des Anges, supérieure des Ursulines de Loudun (Sister Jeanne of the angels, superior of the Ursuline Convent in Loudun)
Théophraste Renaudot (1586-1653), Gilles de la Tourette's hero
Gilles de la Tourette, commentator for la Revue Hebdomadaire, 1892-1900
Correspondence between Octave Lebesgue, known as Georges Montorgueil, and Gilles de la Tourette
Gilles de la Tourette, a poet
Recapitulative list of all Gilles de la Tourette's publications.Digital Access Oxford 2019 - ArticleMandel MA, Mahmoud AA.J Immunol. 1978 Apr;120(4):1375-7.Mutation diabetes in the mouse occurs in the C57BL/Ks strain. All homozygous animals (db/db) develop obesity, elevated blood sugar levels and increased or normal blood insulin concentration. The defects in cellular immunity in db/db mice and their littermate controls were examined both in vivo and in vitro. Significant suppression of delayed footpad swelling and first and second set skin allograft rejection time were observed. In addition, DNA synthesis in spleen cells after nonspecific mitogen stimulation was markedly inhibited. Diabetic animals with a mean blood sugar of 512 +/- 101 mg/100 ml did not respond to exogenous insulin therapy by lowering their blood sugar levels or reversing the defect in DNA synthesis. Adding insulin to spleen cell culture in vitro had no demonstrable effect on their response to mitogens. Thus, mutation diabetic mice with their known defect in the peripheral utilization of insulin have markedly suppressed cell-mediated immune mechanisms.