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  • Book
    Amy K. Reeve, Eve M. Simcox, Michael R. Duchen, Doug M. Turnbull, editors.
    Contents:
    Part I. Mitochondria and Neurodegenerative Disease
    An Introduction to Mitochondria, Their Structure and Functions
    Part II. Mitochondrial Dysfunction: Causes and Effects?
    Neurodegeneration in Mitochondrial Disorders
    The Ageing Brain, Mitochondria and Neurodegeneration
    Mitochondrial Genes and Neurodegenerative Disease
    Mitochondrial Signalling and Neurodegeneration
    Part III. Functional Consequences of Mitochondrial Dysfunction
    Life on the Edge: Determinants of Selective Neuronal Vulnerability in Parkinson's Disease
    Mitochondrial Dynamics and Neurodegeneration
    Mitochondrial Dysfunction and Transport in Demyelinating Disease with Inflammation
    Mitochondria, the Synapse, and Neurodegeneration
    Protein Misfolding and Aggregation: Implications for Mitochondrial Dysfunction and Neurodegeneration
    Mitochondrial Degradation, Autophagy and Neurodegenerative Disease
    The Deleterious Duo of Neurodegeneration: Lysosomes and Mitochondria
    Part IV. Measuring the Contribution of Mitochondrial Dysfunction to Neurodegenerative Disease
    Can We Accurately Model Mitochondrial Dysfunction in Neurodegeneration?
    Mitochondrial Function and Dynamics Imaged In Vivo
    Part V. The Future
    Development of Treatments and Therapies to Target Mitochondrial Dysfunction
    Summary and Conclusions.
    Digital Access Springer 2016
  • Article
    O'Brien AD, Thompson MR, Gemski P, Doctor BP, Formal SB.
    Infect Immun. 1977 Mar;15(3):796-8.
    A toxin extracted from heat-inactivated, alkaline-treated Shigella flexneri 2a showed biological properties similar to those of Shigella dysenteria 1 toxin. The S, flexneri 2a toxin was lethal to mice, enterotoxic for ileal loops of rabbits, and cytotoxic for HeLa cells. A serological relationship between S. flexneri 2a and S. dysenteriae 1 toxin was shown with cross neutralization tests.
    Digital Access Access Options