BookKazunari Kaneko, editor.
Contents:
Part 1. Introduction
1. History of Research on Pathogenesis of Idiopathic Nephrotic Syndrome
Part 2. Minimal-change nephrotic syndrome (MCNS)
2. Hemopexin in Minimal Change Nephrotic Syndrome
3. Angiopoietin-like-4 (Angptl4) in MCNS
4. Co-stimulatory molecule CD80 (B7.1) in MCNS
5. Energy and Mammalian target of rapamycin complex 1 (mTORC1) in minimal change nephrotic syndrome
6. The role of c-mip in the pathogenesis of Minimal Change Nephrotic Syndrome
7. REGULATORY T-CELLS AND OXIDATIVE STRESS IN MINIMAL CHANGE NEPHROPATHY
8. CYTOKINES AS ACTIVE FACTORS IN MINIMAL CHANGE NEPHROTIC SYNDROME
Part 3. Focal segmental glomerulosclerosis (FSGS)
9. Soluble urokinase-type plasminogen activator receptor (suPAR) in Focal Segmental Glomerulosclerosis
10. CYTOKINES AS ACTIVE FACTORS IN FOCAL SEGMENTAL GLOMERULOSCLEROSIS
Part 4. Idiopathic Membranous Nephropathy (IMN)
11. M-Type Phospholipase A2 Receptor (PLA2R) and Thrombospondin Type-1 Domain-Containing 7A (THSD7A) in Membranous Nephropathy
12. Cationic Bovine Serum Albumin as Cause of Membranous Nephropathy: From Mice to Men
Part 5. Treatment in Idiopathic Nephrotic Syndrome
13. Podocytes as a direct target of drugs used in Idiopathic Nephrotic Syndrome.