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  • Book
    Terry F. Davies, editor.
    Contents:
    1: Pituitary
    Overview: The role of medical therapy for secretory pituitary adenomas: current practices and new therapeutics
    Acromegaly, awareness is paramount for early diagnosis: highlights of diagnosis and treatment challenges
    A non-traumatic hip fracture in a young woman: Cushing's disease
    consequences of a late diagnosis and treatment highlights
    Section 2: Thyroid overactivity.- Introduction.- TSH-Secreting Pituitary.- Struma Ovarii.- Exogenous Thyrotoxicosis
    Section 3: Thyroid underactivity.- Thyroid Underactivity: Introduction.- Central Hypothyroidism.- Treatment of hypothyroidism
    Utility of adding T3 (liothyronine).- Hypothyroidism in Pregnancy
    Hypothyroid myopathy and thelogen effluvium
    Section 4: Thyroid cancer.- Introduction on Thyroid Chapters.- Approach to the Patient with an Incidentally Discovered Thyroid Nodule.- Papillary Thyroid Cancer.- Metastatic papillary thyroid cancer.- Management of Metastatic Medullary Thyroid Cancer.- Thyroid cancer and bone metastases
    Section 5: Adrenal.- Primary aldosteronism.- Adrenal Incidentaloma and Subclinical Hypercortisolism.- A Case of Pheochromocytoma
    Section 6: Hyperparathyroidism.- Introduction: Parathyroid Disorders
    Complex hyperparathyroidism.- Differential diagnosis of hypocalcemia.- Management of hypoparathyroidism.- Osteomalacia and primary hyperparathyroidism
    Section 7: Metabolic bone diseases
    Advances and challenges in the management of Osteoporosis.- Multiple Risk Factors for Osteoporosis and Fracture.- Delayed Diagnosis of Osteoporosis.- Misdiagnosis of atypical Femur Fractures
    Section 8: Endocrine Disorders of Men.- Introduction: Endocrine Disorders in Men.- Congenital Hypogonadotropic Hypogonadism.- Klinefelter Syndrome.- Low testosterone and the Metabolic Syndrome
    Section 9: Pregnancy.- Introduction: Endocrine Cases and Pregnancy.- Getting pregnant with PCOS.- Thyrotoxicosis in pregnancy.- Gestational diabetes
    Section 10: Diabetes.- Introduction: Type 2 Diabetes.- Evaluation and Management of the Newly-diagnosed Patient with Type 2 Diabetes.- Transition to Insulin in Patients with Type 2 Diabetes.- Inpatient management
    Section 11: Lipid abnormalities.- Introduction: On Lipids.- LDL-apheresis Therapy for Refractory Familial Hypercholesterolemia.- Familial type II Hyperlipoproteinemia (familial dysbetalipoproteinemia).- Severe hypertriglycideremia
    Section 12: Obesity.- Introduction
    Obesity and the metabolic syndrome.- Managing a metabolic syndrome case.- The Polycystic Ovarian Syndrome (PCOS).- Bariatric Surgery.
    Digital Access Springer 2015
  • Article
    Ojeda SR, Jameson HE, McCann SM.
    Endocrinology. 1977 Feb;100(2):440-51.
    Pituitary LH and FSH repsonses to synthetic LHRH as estimated by increases in plasma FSH and LH 15 and 45 min following its iv injection were enhanced during the first 2 weeks of life, reaching a maximum around day 10-15 and declining thereafter. No AM.-PM. variations in pituitary responsiveness were observed at any age studied. The increased pituitary response found in infantile rats did not appear to be caused by a slower rate of disappearance of LHRH in blood of the younger animals. Ovariectomy-adrenalectomy. (Ovx-Adrx) or Ovx at day 10, but not Adrx alone, resulted in elevated LH and FSH levels 5 days later and almost complete obliteration of the FSH response to LHRH. The LH response was not altered. Treatment with 5alha-dihydrotestosterone (DHT) but not with estradiol benzoate (EB) or testosterone propionate (TP) suppressed the post-Ovx-Adrx rise in plasma LH and FSH. Progesterone (P) potentiated the effect of DHT. Restoration of basal plasma LH and FSH levels (by DHT and/or P) restored FSH responsiveness to exogenous LHRH. EB and TP were ineffective. The LH response was slightly depressed by EB + DHT. It is concluded that the elevated plasma FSH levels in the infantile female rat may be due at least in part to a high degree of pituitary responsiveness to LHRH and/or FSH-RF brought about by steroidal signal of ovarian origin. DHT and P appear to be the steroids responsible for such a stimulatory action.
    Digital Access Access Options