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- BookShabih Zaidi, Mona Nasir.Summary: This book considers the evolution of medical education over the centuries, presents various theories and principles of learning, and discusses different forms of medical curriculum and the strategies employed to develop them, citing examples from medical schools in developed and developing nations. Instructional methodologies and tools for assessment and evaluation are discussed at length and additional elements of modern medical teaching, such as writing skills, communication skills, evidence based medicine, medical ethics, skill labs, and webinars, are fully considered. In discussing these topics, the authors draw upon the personal experience that they have gained in learning, teaching and disseminating knowledge in many parts of the world over the past four decades. .
Contents:
Introduction
Evolution of medical education over millennia
Medical education in twentieth century
Theories and principles of learning :Pedagogy and Andragogy
The triangle of medical education
Curriculum development
Instructional strategies
Assessment and evaluation
Research methodology
Medical writing
Evidence Based Medicine
Communication skills
Medical ethics, etc
Case study and a model of COME ( Community Oriented Medical Education)
Some other models eg. MLEs and curricula in the Caribbean medical schools
Medical education at American University of Barbados
Some other models to view
Epilogue. - OtherContents:
Africa Mandela
Amando en tiempo de guerra
Esta suerte de tenerte
El relevo
Voces del río
Mulukukú
Vuela Fenix
Plegaria
No calles corazón
Solo huellas
Coral y piedra.Print p1987 - ArticleAlarcón-Segovia D, Ruíz-Argüelles A.J Clin Invest. 1978 Dec;62(6):1390-4.Thymus-derived cells with receptors for the Fc portion of immunoglobulin G (Fcgamma+ T cells) have recently been found to have a suppressor function, a function that is decreased in systemic lupus erythematosus (SLE). Fcgamma+ T cells were found significantly diminished in 21 untreated SLE patients, particularly in the 7 patients who had active disease. Most Fcgamma+ T cells were separated with a subpopulation of T cells with low affinity for sheep erythrocytes. Decrease of this subpopulation was dependent on the decrease in Fcgamma+ T cells. Non-T cells with Fcgamma receptors were also diminished in SLE patients, but their decrease did not correlate with disease activity. The decrease in suppressor-cell function in SLE may be a result of loss, rather than of dysfunction, of the suppressor Fcgamma+ T cells.