Live Chat
Today's Hours: 8:00am - 10:00pm
Lane Medical Library

Search

Filter Results

Did You Mean:

Search Results

Sort by
relevance
  • Book
    Mandy L. Corrigan, Arlene A. Escuro, Donald F. Kirby, editors.
    Summary: Handbook of Clinical Nutrition and Stroke is a comprehensive reference on nutrition for the multidisciplinary team caring for stroke patients. Targeting physicians, nurse practitioners, clinical dietitians, and advanced allied health and medical students, this volume provides an introduction on the different types of stroke, associated risk factors, and uniquely featured global perspectives on stroke. In addition to discussing stroke risk factors, the book expands upon treatment and management from the acute care setting through rehabilitation, captures the lifespan of patients affected by stroke.

    Contents:
    Part I: Introduction to Stroke
    Epidemiology of Stroke
    Types of Strokes
    Stroke Risk Factors
    Perspectives and Approach to Stroke Prevention and Therapy
    Part II: Health-Related Risk Factors for Stroke
    Diabetes Mellitus Prevention and Treatment
    Hypertension/Hyperlipidemia/Hyperhomocysteinemia and Nutrition Approaches
    Obesity and Stroke
    Pediatric/Adolescent Stroke
    Stroke in Younger and Older Adults
    Part III: Medical Management of Stroke
    Medical Management of Stroke
    Malnutrition in Stroke
    Fluid and Electrolyte Management
    Nutrition Support
    Enteral Access
    Dysphagia in Stroke
    Stroke Nursing Care
    Stroke Rehabilitation
    Ethical Issues in Stroke Patients
    Suggested Stroke Related Resources for the Practitioner and Patient.
    Digital Access Springer 2013
  • Article
    McDougal JS, Cort SP.
    J Immunol. 1978 Feb;120(2):445-51.
    A sequential culture technique for the in vitro induction and subsequent assay of T helper cells is employed to examine the histocompatibility requirements for antigen recognition by murine T helper cells. F1 T cells are primed in vitro with antigen-pulsed parental strain macrophages and tested for antigen-specific helper activity in cultures containing anti-Thy 1.2 serum and C treated spleen cells from hapten-primed parental or F1 mice. A semiallogenieic system is used and appropriate controls are included to avoid possible complicating effects of allogeneic interactions. The results indicate that F1 T helper cells preferentially stimulate carrier-specific anti-hapten plaque-forming cell responses in spleen cells which are H-2 identical with the macrophage used initially to prime the T cells. Parental spleen cell cultures do not respond to F1 T helper cells which were primed with the other parental strain macrophage. Supplementing this culture with macrophages which are histocompatible with those used to prime the F1T cells is sufficient to restore T helper cell activity. Thus, the genetic restriction described here is between the primed T cell and the macrophage used to elicit secondary responses and not between the T cell and B cell. The results in this semiallogeneic system, however, do not rule out the possibility of additional allogeneic genetic restrictions in the subsequent interaction of T cells with B cells.
    Digital Access Access Options