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  • Book
    Yingze Zhang, editor.
    Summary: This book covers common fracture and dislocation classifications for nearly every part of the human body. For each part, it introduces readers to the historical evolution of fracture classifications, as well as the commonly used classifications, with likelihood from high to low. To describe these classifications, the book combines extensive descriptions, tree and schematic diagrams, elegant drawings and detailed radiological images. This structure will help readers recognize the differences between various classifications, and make proper decisions on the basis of their specific research purposes. As such, the book offers a valuable reference guide for orthopedists, radiologists and medical students alike.

    Contents:
    Classifications of shoulder girdle fractures
    Classifications of humeral fractures
    Classifications of radius and ulna fractures
    Classifications of hand and wrist fractures
    Classifications of dislocation of shoulder and upper limb
    Classifications for spine fractures
    Classifications of pelvic ring fracture and dislocation
    Classifications of femoral fractures
    Classifications of patellar fracture
    Classifications of tibial and fibular fractures
    Classifications of foot fractures
    Classifications of hip joint and lower extremity dislocations
    Classifications of soft-tissue injuries
    Fracture and dislocation classification for children.
    Digital Access Springer 2018
  • Article
    Warner HR, Duncan BK.
    Nature. 1978 Mar 02;272(5648):32-4.
    T4 bacteriophage DNA containing as much as 30% of its thymine replaced by uracil can be synthesised in Escherichia coli deficient in both dUTPase and uracil--DNA glycosidase. This uracil-containing DNA is competent for RNA transcription, and can be packaged into phage which are viable, if the host cells are deficient in uracil--DNA glycosidase activity. If the host cells are not deficient in this glycosidase activity the infecting phage DNA is rapidly attacked, resulting in more than 50% acid-solubilisation of the DNA. The infected cells are inefficiently killed, presumably because of very limited, if any, expression of the phage DNA. These results indicate that this replacement of thymine by uracil in DNA does not seriously impair the biological functionality of T4 DNA, provided the DNA is not subjected to the breakdown (repair) pathway initiated by uracil--DNA glycosidase.
    Digital Access Access Options