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  • Book
    Ayọ̀bámi Adébáyọ̀.
    Summary: "A dazzling story of modern Nigeria and two people caught in the riptides of wealth, power, poverty, and corruption, by the celebrated author of Stay With Me"-- Provided by publisher. Eniola is tall for his age, a boy who looks like a man. Because his father has lost his job, Eniola spends his days running errands for the local tailor, collecting newspapers, begging when he must, dreaming of a big future. Wuraola is a golden girl, the perfect child of a wealthy family. Now an exhausted young doctor in her first year of practice, she is beloved by Kunle, the volatile son of an ascendant politician. When a local politician takes an interest in Eniola and sudden violence shatters a family party, Wuraola and Eniola's lives become intertwined. In her breathtaking second novel, Ayobami Adebayo shines her light on Nigeria, on the gaping divide between the haves and the have-nots, and the shared humanity that lives in between.
    Digital Access
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    Fulltext (1 concurrent user)
    2023 Limited to 1 simultaneous userSUNet ID login required
    Audiobook (1 concurrent user)
    2023 Limited to 1 simultaneous userSUNet ID login required
    Print Access Request
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    New Books Shelf (Duck Room)
    PR9387.9.A319 S64 2023
    1
  • Article
    Herchuelz A, Malaisse WJ.
    J Physiol. 1978 Oct;283:409-24.
    1. The release of 45calcium from prelabelled pancreatic islets is rapidly and almost totally inhibited by lanthanum. 2. Glucose provokes an intitial fall followed by a secondary rise in 45calcium efflux. The latter rise occurs concomitantly with insulin release. Its magnitude is reduced whenever the secretory response to glucose is inhibited, e.g. in the absence of extracellular calcium, presence of Verapamil, or at high magnesium concentration. 3. However, under suitable conditions, the glucose-induced secondary rise in 45calcium efflux is not totally suppressed whilst insulin release is totally abolished. 4. Inversely, when calcium is replaced by barium in the perifusate, glucose increases insulin output without causing any obvious secondary rise in 45calcium efflux. 5. It is concluded that this secondary rise, which originates from a lanthanum-nondisplaceable calcium pool, does not correspond solely to an exocytotic release of 45calcium. It could represent, in part at least, a displacement of 45calcium from cellular sites and reflect a glucose-induced increase in the rate of calcium entry in islet cells.
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