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  • Book
    Michael N. Patlas, Douglas S. Katz, Mariano Scaglione, editors.
    Summary: This reference work provides a comprehensive and modern approach to the imaging of numerous non-traumatic and traumatic emergency conditions affecting the human body. It reviews the latest imaging techniques, related clinical literature, and appropriateness criteria/guidelines, while also discussing current controversies in the imaging of acutely ill patients. The first chapters outline an evidence-based approach to imaging interpretation for patients with acute non-traumatic and traumatic conditions, explain the role of Artificial Intelligence in emergency radiology, and offer guidance on when to consult an interventional radiologist in vascular as well as non-vascular emergencies. The next chapters describe specific applications of Ultrasound, Magnetic Resonance Imaging, radiography, Multi-Detector Computed Tomography (MDCT), and Dual-Energy Computed Tomography for the imaging of common and less common acute brain, spine, thoracic, abdominal, pelvic and musculoskeletal conditions, including the unique challenges of imaging pregnant, bariatric and pediatric patients. Written by a group of leading North American and European Emergency and Trauma Radiology experts, this book will be of value to emergency and general radiologists, to emergency department physicians and related personnel, to obstetricians and gynecologists, to general and trauma surgeons, as well as trainees in all of these specialties.

    Contents:
    Emergency and Trauma Imaging
    Role of Artificial Intelligence in Emergency Radiology
    Vascular Emergencies
    Non-vascular Emergencies
    Imaging of Traumatic Brain Injury
    Imaging of Skull Base Fractures and Complications
    Imaging of Midfacial and Orbital Trauma
    Stroke Imaging
    Imaging of Intracranial Hemorrhage
    Imaging of Brain Infections.
    Digital Access Springer 2022
  • Article
    Sumrada R, Cooper TG.
    J Bacteriol. 1978 Oct;136(1):234-46.
    Saccharomyces cerevisiae responds to deperivation of nutrients by arresting cell division at the unbudded G1 stage. Cells situated outside of G1 at the time of deperivation complete the cell cycle before arresting. This prompted an investigation of the source of nutrients used by these cells to complete division and the mechanisms controlling their availability. We found a close correlation between accumulation of unbudded cells and loss of previously formed allophanate hydrolase activity after nutrient starvation. These losses were not specific to the allantoin, system since they have been observed for a number of other enzymes and also when cellular protein levels were monitored with [3H]leucine. Loss of hydrolase activity was also observed when protein synthesis was inhibited either by addition of inhibitors or loss of the prtl gene product. We found that onset of nutrient starvation brought about release of large quantities of arginine and allantoin normally sequestered in the cell vacuole. Treatment of a cells with alpha-factor resulted in both the release of allantoin and arginine from the cell vacuole and the onset of intracellular protein degradation. These effects were not observed when either alpha cells or a/alpha diploid strains were treated with alpha-factor. These data suggest that release of vacuolar constitutents and protein turnover may be regulated by the G1 arrest signal.
    Digital Access Access Options