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- BookSumita Mehta, Anshul Grover, editors.Summary: This book is a complete guide to the diagnosis and management of any infectious disease which may affect the mother or the fetus during pregnancy. Pregnancy is a unique condition in which the interplay of endocrine and immune influences leads to altered severity and susceptibility to infectious diseases. These infections, in turn, are a substantial cause of maternal and perinatal morbidity. The book discusses the immunologic, clinical and epidemiologic evidence for altered responses during pregnancy. Several infections have unique consequences in pregnancy. Some infections have vertical transmission, and their management focuses on decreasing perinatal transmission. Others can be transmitted transplacentally and cause congenital infection. While still, other common infections like gastroenteritis, UTI, tuberculosis, leprosy or certain dermatological and oral conditions can cause pregnancy complications. This book discusses all such diseases in detail as well as suggests means for early identification and appropriate treatment for them. A separate chapter adequately covers the novel coronavirus infection associated with management challenges in pregnant women. The book includes dedicated sections on postpartum infections and fetal outcomes associated with maternal infections. It reviews strategies to prevent infection in obstetrics that plays a key role in decreasing the global burden of maternal morbidity and mortality. The book is relevant for practicing obstetricians and gynecologists, post-graduate students of obstetrics and gynecology as well as general practitioners, family medicine specialists, primary health care workers and undergraduate medical students.
Contents:
Part I: Epidemiology and Physiology of Infections in Pregnancy
Changing Epidemiology of Infections in Pregnancy
Physiological and Immunological Changes in Pregnancy Predisposing to Infections
Microflora of the Genital Tract
Part II: Infections in Pregnancy
Respiratory Infections in Pregnancy
Viral Infections in Pregnancy Presenting as Rash
Protozoal & Helminthic Infections in Pregnancy
Vector Borne Infections in Pregnant Women
Sexually Transmitted Diseases Affecting Pregnancy
HIV in Pregnancy
Vulvovaginitis in Pregnancy
Urinary Tract Infection During Pregnancy
Group B Streptococcus Infection & MRSA Affecting Pregnancy
Hepatitis in Pregnancy
Enterovirus Infections in Pregnancy
Leprosy in Pregnancy
Gastroenteritis in Pregnancy
Part III: Miscellaneous Infections
Surgical Conditions-Appendicitis & Cholecystitis During Pregnancy
Dental Health in Pregnancy.-Animal Bite and Rabies Infection
Tetanus and its Prevention in Pregnancy
Part IV: Puerperal Infections
Puerperal Sepsis
Puerperal Mastitis and Breast Abscess
Surgical Site Infection and Prevention
Part V: Infections & Feto-maternal Outcomes
Abortion & Infections: Cause & Effect
Fetal Manifestations of Infections in Pregnancy
Infections as a Cause of Preterm Labor
Part VI: Infection Prevention Practices
27. Intrauterine Infections- Diagnosis and Management
Vaccination in Pregnancy
Infection Prevention Practices During Delivery
Rational Use of Antibiotics in Pregnancy. - ArticleRoth EF, Friedman M, Ueda Y, Tellez I, Trager W, Nagel RL.Science. 1978 Nov 10;202(4368):650-2.The kinetics of sickling of malaria-infected red cells from humans with sickle cell trait were studied in vitro in an attempt to obtain direct experimental evidence for a selective advantage of the hemoglobin S heterozygote in a malarious region. The sickling rates of cells infected with Plasmodium falciparum and of non-infected cells were studied both in the total absence of oxygen (by dithionite addition) and at several different concentrations of oxyhemoglobin which might obtain in vivo. In all cases, red cells containing small plasmodium parasite forms (ring forms) sickled approximately eight times as readily as uninfected cells. Cells containing large parasitic forms (trophozoites and schizonts) appeared to sickle less readily than uninfected cells, by light microscopy criteria, but electron micrographs demonstrated the presence of polymerized deoxyhemoglobin S with a high frequency. It is concluded that enhanced sickling of plasmodium-infected AS cells may be one mechanism whereby the hemoglobin S polymorphism is balanced in favor of the heterozygote.