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  • Book
    by Peter H. Gilligan, Ph.D., Director, Clinical Microbiology-Immunology Laboratories, ... Show More University of North Carolina Hospitals, Professor, Microbiology-Immunology and Pathology, University of North Carolina School of Medicine Chapel Hill, North Carolina ; Daniel S. Shapiro, M.D., Professor and H. Edward Manville, Jr. Endowed Chair of Internal Medicine, Department of Internal Medicine - Reno, University of Nevada School of Medicine, Reno, Nevada ; Melissa B. Miller, Ph. D., Director, Clinical Molecular Microbiology Laboratory, Associate Director, Clinical Microbiology-Immunology Laboratories, University of North Carolina Health Care, Associate Professor, Pathology and Laboratory Medicine, University of North Carolina School of Medicine, Chapel Hill, North Carolina.
    Summary: This book challenges students to develop a working knowledge of the variety of microorganisms that cause infections in humans. This valuable, interactive text will help them better understand the clinical importance of the basic science concepts presented in medical microbiology or infectious disease courses.

    Contents:
    Urogenital tract infections
    Respiratory tract infections
    Gastrointestinal tract infections
    Skin & soft tissue infections
    Central nervous system infections
    Systemic infections
    Advanced cases.
    Digital Access Wiley 2014
  • Article
    Price P.
    Immunology. 1978 Sep;35(3):531-7.
    Plaque-forming cell responses and antibody titres to sheep red cells (SRC) were elevated in normally-fed irradiated adult mice reconstituted with spleen cells from young mice which had received a 4% albumin diet, compared with those given cells from normally-fed littermates. This was independent of the dose of cells transferred (5 x 10(6)--4 x 10(7)), the day of assay (3--10) and the duration of protein-deficiency (1 or 3 weeks). It suggests that the depressed responses produced by protein-deficient (donor) mice reported in earlier papers resulted from an impairment of reticuloendothelial function or a shortage of protein reserves, not directly associated with the spleen. As protein-deficiency (of the donor) accelerated the initiation of the adoptive response and increased the cell yields of the recipients it is considered likely that an enhancement of cell proliferation during the first 3--5 days after transfer was responsible for the elevated responses.
    Digital Access Access Options