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  • Book
    Kursad Turksen, editor.
    Contents:
    Recent Advances in the Generation of [beta]-cells from Induced Pluripotent Stem Cells As a Potential Cure for Diabetes Mellitus
    Differentiated cells derived from hematopoietic stem cells and their applications in translational medicine
    Organoids in Tissue Transplantation
    Aldo keto reductases AKR1B1 and AKR1B10 in cancer: molecular mechanisms and signaling networks
    Bilayer Scaffolds for Interface Tissue Engineering and regenerative medicine: A Systematic Review
    Pluripotent Stem Cell Derived Neurons as In Vitro Models for Studying Autosomal Recessive Parkinson's Disease (ARPD): PLA2G6 and Other Gene Loci
    Stem Cell Applications In Lysosomal Storage Disorders: Progress And Ongoing Challenges
    Mechanisms of Drug Resistance and Use of Nanoparticle Delivery to Overcome Resistance in Breast Cancers
    Telomere length and oxidative stress in patients with St-elevation and Non-St-elevation myocardial infarction
    Eosinophils as major player in Type 2 inflammation: autoimmunity and beyond.
    Digital Access Springer 2021
  • Journal
    Print 1- ; 1965.
  • Article
    Skrabanek P, Powell D.
    Clin Endocrinol (Oxf). 1978 Aug;9(2):141-54.
    The concept of ectopic insulin production is challenged on the basis of a review of 120 cases from the literature on extrapancreatic tumours associated with hypoglycaemia in which insulin or insulin-like activity were measured. No case met two or more of five criteria of ectopic hormone production. The evidence indicates that hypoglycaemia of extrapancreatic tumours cannot be attributed to insulin. In those rare cases in which plasma insulin was reported as high, pancreatic beta-cells could not be excluded as the source of insulin. Interestingly, many of these dubious cases had carcinoid histology. The review also points out a close association between some spindle-cell tumours and carcinoid tumours which may be relevant to discussion on the disputed origin of some "mesothelial" tumours. Nonsuppressible insulin-like activity (NSILA) consists of a number of factors mimicking insulin activity which compete with insulin or proinsulin for membrane receptors and may crossreact in bioassays, immunoassays, and receptor assays. The question of whether one or several of these substances may be responsible for extrapancreatic hypoglycaemia remains to be elucidated.
    Digital Access Access Options