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  • Book
    John P. Mulhall, Mario Maggi, Landon Trost, editors.
    Summary: "This book aims to address a number of controversies concerning the use of testosterone treatment. It explains topics that clinicians regularly encounter such as whether to use free or total testosterone in the evaluation of the patient with testosterone deficiency; what factors actually impact testosterone levels, polycythemia, gynecomastia, bone density, and hyperprolactinemia in the testosterone deficient patient; critical analysis of the numerous questionnaires that are available to clinicians; and the impact of testosterone therapy on male fertility, cardiovascular disease, and prostate events including prostate cancer. Guiding the reader in both evaluation and management, the book also illuminates novel concepts in the T space such as testosterone use in the transgender population, T therapy as an endothelial modulator, bipolar testosterone therapy in the patient with advanced prostate cancer, and testosterone therapy as a performance enhancer.Controversies in Testosterone Deficiency is intended for any clinician involved in the care of patients with testosterone deficiency, exploring hot topics and correcting existent misinformation in the routine care of patients."--publisher's description.

    Contents:
    What to Measure: Testosterone or Free Testosterone?
    Erythrocytosis in Patients on Testosterone Therapy
    Role of Gonadotropins in Adult-Onset Functional Hypogonadism
    Hyperprolactinemia in the Man with Testosterone Deficiency
    Testosterone and Disordered Sleep
    Testosterone Therapy and Male Fertility
    Testosterone and Prostate Effects
    Testosterone in Females
    Testosterone in Transgender Population
    Testosterone as a Performance Enhancer.
    Digital Access Springer 2021
  • Article
    Liston AJ, Baker JR.
    J Gen Microbiol. 1978 Aug;107(2):253-62.
    Observations by phase contrast, fluorescence and electron microscopy showed that epimastigotes of Trypanosoma (Schizotrypanum) dionisii (grown in vitro) were phagocytosed posterior end first by mouse peritoneal macrophages in vitro. Many were subsequently digested as a result of phagosome-lysosome fusion but others survived by apparently inhibiting this fusion and/or escaping from the phagosome into the host cell's cytoplasm. These survivors replicated as amastigotes. Long trypomastigotes, separated from populations grown in vitro by passage down a column of glass beads (with or without prior exposure to guinea-pig serum), were phagocytosed by either pole and all were subsequently digested.
    Digital Access Access Options