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  • Book
    Stefano Fanti, Gopinath Gnanasegaran, Ignasi Carrió, editors.
    Summary: This text atlas is a superb guide to the use of PET-CT for the evaluation of treatment response in oncology patients based on its ability to assess tumor metabolic status. The first part of the book explains the role of PET-CT in response evaluation in different treatment settings. For comparison, overviews of the value and limitations of CT alone, PET alone, and anatomical and functional MRI are included. Guidance is also provided on the reporting of PET-CT scans in post-therapy scenarios. The second part of the book describes and illustrates the use of PET-CT with FDG and other tracers to assess the treatment response of malignancies at different anatomic sites. Featuring a wealth of images, informative case-based discussion, and evidence-based teaching points, these disease-specific chapters clearly demonstrate the key role that PET-CT can play in distinguishing early responders from patients who are non-responders or are resistant to treatment. Prompt and accurate evaluation of treatment response is vital as we enter the era of individualized medicine, and this atlas will persuade readers of the considerable advantages of PET-CT over conventional radiological and clinical methods.

    Contents:
    PART I General Chapters: Introduction to Treatment response evaluation: Science and Practice
    CT in Treatment response evaluation Overview
    MRI & Diffusion weighted MRI in Treatment response evaluation- Overview
    PET & PET-CT in Treatment response evaluation-Overview
    Conventional Radiological Techniques and PET-CT in Treatment response evaluation in post-surgical setting
    Conventional Radiological Techniques and PET-CT in Treatment response evaluation in chemotherapy setting
    Conventional Radiological Techniques and PET-CT in Treatment response evaluation in radiation oncology
    Conventional Radiological Techniques and PET-CT in Treatment response evaluation in Immunotherapy settings
    Treatment response evaluation of bone metastases using 18F-NaF
    How to report PET-CT scans in post therapy scenarios: Do's and don'ts
    PART II Atlas Articles: 18F-FDG & Non FDG PET-CT in Treatment response evaluation in Head and neck cancer
    18F-FDG & Non FDG PET-CT in Treatment response evaluation in lung cancer
    18F-FDG & Non FDG PET-CT in Treatment response evaluation in neuro-oncology
    18F-FDG & Non FDG PET-CT in Treatment response evaluation in hepatobiliary cancer
    18F-FDG & Non FDG PET-CT in Treatment response evaluation in gastroesophageal cancer
    18F-FDG & Non FDG PET-CT in Treatment response evaluation in Lymphoma and Non-hodgkins lymphoma
    18F-FDG & Non FDG PET-CT in Treatment response evaluation in breast cancer
    18F-FDG, 18F-Choline & 68Ga-PSMA PET-CT in Treatment response evaluation in prostate cancer
    18F-FDG & Non FDG PET-CT in Treatment response evaluation in gynaecological cancers
    18F-FDG & Non FDG PET-CT in Treatment response evaluation in colorectal cancer
    18F-FDG & Non FDG PET-CT in Treatment response evaluation in soft tissue sarcomas
    FDG & Non FDG PET-CT in Treatment response evaluation in malignant melanoma
    18F-FDG & Non FDG PET-CT in Treatment response evaluation in myeloma
    FDG PET-CT & 18F-NaF in Treatment response evaluation in bone metastases and bone tumours
    18F-FDG & Non FDG PET-CT in Assessment of treatment response in paediatric oncology
    68Ga-DOTA PET-CT in Treatment response evaluation in NETs
    18F-DOPA PET-CT in Treatment response evaluation
    18F-FLT PET-CT in Treatment response evaluation
    Brain PET-CT in Treatment response evaluation.
    Digital Access Springer 2021
  • Article
    Tittle TV, Rittenberg MB.
    J Immunol. 1978 Sep;121(3):936-41.
    Spleen cells from mice primed with the thymus-dependent antigen trinitrophenyl keyhold limpet hemocyanin several months earlier can be cultured in vitro to give vigorous IgG antihapten PFC responses to thymus-dependent (TD) and thymus-independent (TI) forms of the same hapten. Here we show that the IgG memory precursors that respond to these two forms of the hapten constitute functionally distinct subpopulations. We have designated these subpopulations as B1gamma and B2gamma to represent secondary precursor cells responding to TI and TD antigens, respectively. Three types of evidence for these subpopulations are presented: 1) In vitro secondary IgG responses to TD and TI forms of the TNP hapten are additive when both forms are added to the same culture. 2) The precursor frequencies for the TD and TI antigens are additive, but addition is not observed between two TD or two TI antigens. 3) Each population can be selectively eliminated by BUdR and light treatment without affecting the other population. The ontogenetic relationships between these subpopulations are discussed in relation to all presently proposed subpopulations B1mu, B2mu, B1gamma, and B2gamma.
    Digital Access Access Options