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  • Book
    Savyasachi Thakkar, Erik A. Hasenboehler, editors.
    Summary: The incidence and prevalence of post-traumatic arthritis (PTA) is increasing globally due to longevity of life and increased activity and injuries. Compared to the non-traumatic etiology of arthritis, post-traumatic arthritis differs in presentation, severity and complexity of management. This book illustrates all pertinent facets of post-traumatic arthritis, from the basic science and diagnostic modalities to their management and outcomes. The opening section reviews the current scientific literature on PTA and presents the preferred imaging techniques and interpretation methods. A unique chapter on the economic impact of PTA is also included here. The subsequent two sections discuss both the upper and lower extremity, respectively, with each joint covered in a dedicated chapter of its own, describing the mechanisms of injury, assessment, management strategies and outcomes. Case material provides real-world context for the concepts and techniques presented, supported by generous radiology and intraoperative photos and figures. With Post-Traumatic Arthritis, orthopedic surgeons, as well as trauma surgeons, rheumatologists and associated clinical staff, will receive a comprehensive framework on which to base their clinical decisions and enhance their post-operative outcomes.

    Contents:
    Chapter 2: Imaging Modalities for Post-traumatic Arthritis
    Introduction
    Conventional Radiography
    MR Imaging
    Association Between Pain and Imaging Findings of OA
    References
    Chapter 3: Economic Implications of Post-traumatic Arthritis of the Hip and Knee
    Introduction
    Post-traumatic Osteoarthritis of the Knee
    Types of Injuries Associated with PTOA of the Knee
    Anterior Cruciate Ligament Injuries
    Meniscus Injuries
    Intra-articular Fractures
    Management of Post-traumatic Osteoarthritis of the Knee
    Primary Prevention
    Secondary Prevention
    Tertiary Prevention Post-traumatic Osteoarthritis of the Hip
    Types of Injuries Associated with PTOA of the Hip
    Hip/Acetabular Fractures
    Hip Dislocations and Osteonecrosis
    Management of PTOA in the Hip
    Primary Prevention
    Secondary Prevention
    Tertiary Prevention
    Costs Associated with Post-traumatic Osteoarthritis
    Recommendations
    Summary
    References
    Part II: Post-traumatic Arthritis of the Upper Extremity
    Chapter 4: Post-traumatic Glenohumeral Arthritis
    Introduction
    Causes
    Fractures
    Instability
    Other Causes
    Treatment
    Nonoperative Treatment Elbow Arthrodesis
    Conclusion
    References
    Chapter 6: Post-traumatic Arthritis of the Wrist
    Introduction
    Main Text
    Wrist Anatomy
    Carpus and Intercarpal Joints
    Distal Radioulnar Joint
    Wrist Arthritis
    Evaluation
    Intercarpal and Radiocarpal Arthritis
    Scapholunate Advanced Collapse (SLAC) and Scaphoid Nonunion Advanced Collapse (SNAC)
    Management
    S4CF Versus PRC
    Wrist Denervation
    Total Wrist Arthroplasty and Arthrodesis
    Surgical Technique
    Isolated Radiocarpal Arthritis
    Management
    Isolated Intercarpal Arthritis
    Digital Access Springer 2021
  • Book
    Digital Access Full text via HathiTrust, v. 1-
  • Article
    Bresler SE, Noskin LA, Stepanova IM, Kuzovleva NA.
    Mol Gen Genet. 1978 Jul 06;163(1):75-85.
    The effect of radioprotection of indolylalkylamines (5-methoxytryptamine) and aminothiols (cysteamine) on E. coli cells is practically absent if the cells have genetic defects in the repair systems. This means that the explanation of radioprotection by scavenging of free radicals is invalid and that specific repair mechanisms may be involved. In order to explain the radioprotective mechanism it was suggested that the radioprotectors interact with the damaged sites in DNA so that they become partly screened from repairing endonucleases. Under these conditions the reduction of incision rate results in diminished enzymatic induction of lethal double-strand breaks in DNA, this being important only in wild type cells. To prove this hypothesis an experimental procedure was developed using bacterial cells carrying plasmids (ColE1). This procedure enabled to determine the in vivo rate of enzymatic incision of gamma-sites. It was found that the protectors did not change the total amount of gamma-damages in DNA but reduced the rate of enzymatic incision.
    Digital Access Access Options