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- BookMartin Lacher, Shawn D. St. Peter, Augusto Zani, editors.Summary: Providing core information on pediatric surgery, this book serves as a supplement to standard pediatric surgical textbooks. It offers pearls of wisdom that will help those who participate in pediatric surgical care, as well as to provide state-of-the-art insights based on physiological principles, literature reviews, and clinical experience. This book is an ideal tool to help readers prepare for questions they will be asked on ward rounds, in the OR, or in oral exams. The depth of exploration is intended for medical students, residents in pediatrics and pediatric surgery, pediatric surgical trainees, pediatric nurse practitioners, primary care pediatricians, and family practitioners.
Contents:
Intro
Foreword by Benno Ure
Foreword by George W. Holcomb
Foreword by Agostino Pierro
Preface
Contents
Chapter 1 Evaluation of the Pediatric Surgical Patient
Abstract
1.1 Introduction
References
Chapter 2 Nutrition, Fluids and Electrolytes for the Pediatric Surgical Patient
Abstract
References
Chapter 3 Chest Wall Deformities
Abstract
Bibliography
Chapter 4 Congenital Diaphragmatic Hernia
Abstract
References
Chapter 5 Esophageal Atresia With or Without Tracheoesophageal Fistula
Abstract
References Chapter 6 Gastroesophageal Reflux Disease
Abstract
References
Chapter 7 Caustic Ingestion of the Esophagus
Abstract
References
Chapter 8 Esophageal Foreign Bodies
Abstract
References
Chapter 9 Congenital Lung Malformations
Abstract
References
Chapter 10 Acquired Lung and Pleural Disease
Abstract
References
Chapter 11 Tracheobronchial Foreign Bodies
Abstract
References
Chapter 12 Pediatric Surgical Diseases of the Larynx, Trachea, and Bronchi
Abstract
References
Chapter 13 Mediastinal Masses
Abstract
References Chapter 14 Congenital Cardiac Anomalies
Abstract
References
Chapter 15 Hemangiomas and Vascular Malformations
Abstract
References
Chapter 16 Umbilical Problems
Abstract
References
Chapter 17 Hernias-Epigastric, Inguinal and Incisional
Abstract
17.1 Inguinal Hernia
17.2 Epigastric Hernia
17.3 Incisional Hernia
References
Chapter 18 Abdominal Wall Defects, Gastroschisis and Omphalocele
Abstract
References
Chapter 19 Exstrophy-Epispadias Complex
Abstract
References
Chapter 20 Prune Belly Syndrome
Abstract
References Chapter 21 Normal Embryology, Anatomy, and Physiology of the Gastrointestinal Tract
Abstract
References
Chapter 22 Small Intestinal Obstruction
Abstract
References
Chapter 23 Intestinal Atresia and Webs
Abstract
References
Chapter 24 Malrotation and Midgut Volvulus
Abstract
References
Chapter 25 Gastrointestinal Surgical Aspects of Cystic Fibrosis
Abstract
References
Chapter 26 Necrotizing Enterocolitis (NEC)
Abstract
References
Chapter 27 Meckel's Diverticulum & Vitelline Duct Remnants
Abstract
References
Chapter 28 Appendicitis Abstract
References
Chapter 29 Intussusception
Abstract
References
Chapter 30 Small Left Colon Syndrome (SLCS)
Abstract
References
Chapter 31 Disorders of Colonic Motility/Hirschsprung Disease
Abstract
References
Chapter 32 Inflammatory Bowel Disease
Abstract
References
Chapter 33 Gastrointestinal Bleeding
Abstract
References
Chapter 34 Anorectal Malformations
Abstract
References
Chapter 35 Anorectal Complaints (Proctology): Hemorrhoids, Fissures, Abscesses, Fistulae
Abstract
35.1 Hemorrhoids
35.2 Anal Fissures
35.3 Abscesses - ArticleRousseaux-Prevost R, Capron M, Bazin H, Capron A.Immunology. 1978 Jul;35(1):33-9.Parasite specific IgE antibodies in rats infected with Schistosoma manoni were measured by passive cutaneous anaphylaxis (PCA) reactions and by the technique of immuno-adsorption. Two strains, one a low IgE producer (Fischer rats) and the other a high IgE producer (Hooded-Lister rats) were studied. In Fischer rats, a time course study of the occurrence of IgE antibodies and resistance to reinfection was made. Parasite specific IgE levels measured by immuno-adsorpiton were much lower than total IgE levels and a similar percentage of specific IgE (about 8%) was in the two strains.IgE antibodies were maximum at day 30 and day 60 after infection; however, a third peak at day 90 was observed only in Fischer rats. Some discrepancies between results obtained by PCA and immunosorbent techniques have been observed, which could be explained by differences in the affinity of IgE antibodies during infection or by the presence of total IgE in the PCA assay. There was a close parallelism between specific IgE antibodies levels and the course of immunity in Fischer rats. This parallelism supports the view that IgE could play a pre-eminent role in protective immunity in rat schistosomiasis.