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  • Book
    Paul M.W. Hackett.
    Summary: This book presents the facet theoretical framework as a tool for facilitating the conception of complex animal behaviour research and the design of research procedures through employing mapping sentences. Using the facet theoretical framework, this book takes a holistic view of bird behaviour. Components of bird behavior are identified and then reassembled to facilitate an understanding of the behaviour in the context of its natural occurrence. This provides new insight on both the parts of the behaviour and how these interact as a whole. The multi-faceted approach to designing, evaluating and understanding bird behavior presented offers a template that is adaptable for investigating a wide variety of avian species and different forms of behaviour. Behavioural biologists, animal and comparative psychologists, other natural and behavioural scientists, as well as students of these disciplines will find this book to be an interesting and enlightening read.

    Contents:
    Preface
    Chapter 1. Studying Birds
    Chapter 2. Mapping Sentences and Facet Theory
    Chapter 3. How to Develop a Mapping Sentence: The Example of Avian Sleep
    Chapter 4. Complex Avian Behaviour and Cognition: A Mapping Sentence Approach
    Chapter 5
    Studying Avian Cognition in the Wild: Avian Psychometrics
    Chapter 6. Interpreting Avian Play, Murmuration of Starlings and Oology (Classifying Birds' Eggs)
    Chapter 7. Conclusion
    Glossary
    Index.
    Digital Access Springer 2020
  • Article
    Pliskin ME, Prehn RT.
    Transplantation. 1978 Jul;26(1):19-24.
    The hypothesis tested was that tumor-specific transplantation antigens of chemically induced tumors cross-react with allogeneic histocompatibility antigens. This hypothesis makes several predictions that can be tested experimentally. First, tumors should grow better and be less immunogenic in certain F1 hybrids than in their syngeneic parents, owing to the hypothecated cross-reactivity of the tumor-specific transplantation antigens with F1 antigens. This is in contrast to the more common observation that parental strain tumors grow worse in the F1 hybrids than they do in the parent. Also certain allogeneic skin grafts might immunize the parental strain mice against their syngeneic tumors, and, finally, immunizing parental mice with syngeneic tumor might cause accelerated rejection of certain skin allografts. The results show that certain tumors grew better in the F1 mice than they did in the parents but that the tumors were not less immunogenic in the F1 hybrids. Mice immunized against alloantigens showed a dose-dependent enhancement of syngeneic tumor growth. Finally, mice immunized with syngeneic tumors demonstrated an apparent prolongation of certain skin allografts. The discussion considers possible alternatives explaining these results.
    Digital Access Access Options