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  • Book
    symposium organizers and proceedings editors, Terri Grodzicker, David Stewart and Bruce Stillman.
    Summary: Summary: Order and Disorder in Brains and Behavior. "Topics addressed at the 2019 Symposium included RNA-based structures; RNA modifications; nuclear localization of RNA;quality control and editing; RNA and gene regulation; cotranscriptional splicing; intron-exon boundaries;alternative polyadenylation; transposon control; small noncoding RNAs; long noncoding RNAs; RNA anddevelopment; membraneless organelles; phase separation; RNA-based diseases; and novel RNA functions."--Foreword.

    Contents:
    Dorcas Cummings Lecture: Dorcas Cummings Lecture.
    Digital Access Cold Spring Harbor Lab Press 2020
  • Article
    Bara L, Samama M, Bilski-Pasquier G, Bloch MF, Rochas S.
    Pathol Biol (Paris). 1977 Dec;25 Suppl:48-54.
    Our aim in this work has been to obtain precisions on the bleeding mechanism during fibrinolysis induced by streptokinase in the rabbit. Haemostasis was studied in vivo by the method oh Honour and Russell. The injection of 40 000 u/kg of streptokinase in 10 minutes induces rapidly fibrinolysis, which lasts for approximately one hour which is not accompanied by changes either in coagulation factors, or in platelet function. In vivo : . the adhesion of platelets to the vascular lesion is normal; . platelet aggregation is normal as is the formation of intravascular thrombi; . on the other hand the intra mural platelet thrombus is unstable. The embolization of the intravascular thrombi is responsible for new hemorrhages which can be correlated with the intensity of fibrinolysis. Thus, even in the absence of fibrinogenolysis, of fibrinogen degradation products FPD, of abnormal thrombin generation and of altered platelet function, the fibrinolytic activity induced by streptokinase could alone be responsible for bleeding episodes, occuring during thrombolytic treatment by streptokinase in man.
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