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  • Book
    edited by Jeffrey C. Gershel, Ellen F. Crain ; associate editor, Sandra J. Cunningham ; assistant editor, James A. Meltzer.
    Summary: "Extensively updated, with chapter revisions and an array of new material, the Sixth Edition of this Clinical Manual offers an indispensable resource on Emergency Pediatrics. Incorporating the latest guidelines concerning a wide range of conditions, the text is an up to date and practical resource for use at the point of care. Providing critical information in all sites where sick and injured children receive treatment - from emergency departments to private offices and primary care settings - this Sixth Edition enables first-rate care. Fully updated with current knowledge and guidance within the field. New topics include Ovarian Emergencies, Bedside Ultrasound, Zika Virus and Commercial Sexual Exploitation among others. Clear guidance enables effective patient evaluation and follow-up. Streamlined chapters, with increased use of tables to enable readers to locate critical information rapidly. Successful and trusted for more than 30 years, this updated handbook is a key resource for pediatricians, emergency medicine physicians, family practitioners, and trainees"--Provided by publisher.
    Digital Access Cambridge 2018
  • Article
    König W, Tesch H, Frickhofen N.
    Eur J Immunol. 1978 Jun;8(6):434-7.
    This study describes the generation and release of an eosinophil chemotactic factor from human polymorphonuclear neutrophils, rat basophilic leukemia cells, and from a lymphocyte monocyte basophil suspension by arachidonic acid (AA). The eosinophil chemotactic factor (ECF) is highly specific for eosinophils and resembles the ECF activity obtained from human polymorphonuclear neutrophils after stimulation with the Ca ionophore or during phagocytosis. In this regard, AA-induced ECF represents a biological activity distinct from oxidized AA and its conversion products. AA may therefore have a dual function: it represents an important mechanism of cell activation; as AA is converted into prostaglandins, it appears likely that they exert a modulatory and a suppressive role on biological functions, such as chemotaxis and phagocytosis.
    Digital Access Access Options