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  • Book
    Irini Sereti, Gregory P. Bisson, Graeme Meintjes, editors.
    Summary: Globally, HIV-associated tuberculosis is one of the most important causes of infection-related death, accounting for over 300,000 deaths worldwide in 2017. The HIV epidemic has undermined TB control efforts in many countries across the world, as the virus has important modifying effects on the pathogenesis, diagnosis, and treatment of TB. The management of HIV-associated TB is also complicated by rapid clinical progression, immune reconstitution inflammatory syndrome, drug-drug interactions, and shared toxicities. The past two decades have yielded thousands of research publications and review articles on HIV-associated TB. HIV and Tuberculosis: A Formidable Alliance consolidates this massive amount of data into a single resource. With contributions from myriad disciplines, including epidemiology, immunology, public health, and clinical medicine, this book provides well-rounded and thorough coverage that will appeal to researchers and clinicians alike.

    Contents:
    1. Overview of the HIV-associated tuberculosis epidemic
    2 . Epidemiology of Drug-susceptible, Drug-resistant Tuberculosis and HIV in Africa
    3. Modelling the HIV-associated TB epidemic and the impact of interventions aimed at epidemic control
    4. Immune responses to Mycobacterium tuberculosis and the impact of HIV infection
    5. Clinical Manifestations of HIV-associated tuberculosis in adults
    6. The tuberculosis-associated immune reconstitution inflammatory syndrome (TB-IRIS)
    7. Diagnosis of HIV-associated tuberculosis
    8. Recent advances in the treatment of latent tuberculosis infection among adults living with HIV
    9. Treatment of drug-sensitive tuberculosis in persons with HIV
    10. Drug-resistant TB and HIV
    11. Co-treatment of tuberculosis and HIV: pharmacologic considerations
    12. HIV and TB in children
    13. Neurological TB in HIV.
    Digital Access Springer 2019
  • Article
    Jørgensen ST.
    Antimicrob Agents Chemother. 1978 May;13(5):710-5.
    The plasmids in 19 chloramphenicol-resistant Escherichia coli strains of three pig pathogenic antigen types were studied in conjugation and transduction experiments. The plasmids had identical resistance patterns: streptomycin, spectinomycin, sulfonamides, and chloramphenicol (Sm, Sp, Su, Cm) and belonged to IncFII. One plasmid carried ampicillin resistance in addition. Restriction enzyme analysis of the deoxyribonucleic acid from five of the plasmids originating from the same herd showed that their digestion patterns with EcoRI were indistinguishable. EcoRI cleaved the deoxyribonucleic acid of a sixth plasmid from the same herd and displayed nine of the ten bands of the other five plasmids plus an additional six. It appears that the five plasmids with identical restriction patterns have a common origin and may be copies of the same plasmid from which the sixth may have developed. Four strains carried two plasmids each. In two of these strains, a plasmid with a tetracycline marker (Tc), or possibly the tetracycline marker alone, recombined frequently with the Sm Sp Su Cm plasmid without destroying any known function of the latter. The possibility that Tc is carried on a translocation sequence is discussed.
    Digital Access Access Options