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  • Book
    Lizhi Zhang, Vishal S. Chandan, Tsung-Teh Wu, editors.
    Summary: This book covers the entire spectrum of non-neoplastic diseases within the gastrointestinal (GI) tract including new entities, recent developments, and questions that arise in the practice of GI pathology. The text is organized by specific organ site, with related disorders of each organ site subgrouped into several chapters based on their common features. Pathologists can quickly find the disorders they are interested and review similar disorders in the same chapter to help them make a correct diagnosis. Each major and common entity is described in detail with its definition, clinical features, pathological features (covering both the gross and microscopic details), differential diagnosis and treatment/prognosis. All chapters also highlight the use of special/immunohistochemical stains and other supporting studies as needed with a focus on providing a practical differential diagnosis rather than just a list of potential associations. This book is extensively illustrated with both gross and microscopic images that act as an integral part of the information provided in the text. Written by practicing GI/liver pathologists and gastroenterologists from the Mayo Clinic, Surgical Pathology of Non-neoplastic Gastrointestinal Diseases serves as a comprehensive yet practical guide for diagnostic surgical pathologists with a passion for GI pathology. -- Provided by publisher.

    Contents:
    Part I: Introduction: 1. Normal histology of gastrointestinal tract / Vishal S. Chandan
    2. Endoscopy, tissue processing, stains, and special tests / Ferga C. Gleeson and Lizhi Zhang. Part II: Non-neoplastic diseases of the esophagus: 3. Reflux esophagitis and Barrett esophagus / Jason T. Lewis
    4. Eosinophilic esophagitis / Thomas C. Smyrk
    5. Drug-induced injury, injections, and congenital and miscellaneous disorders / Taofic Mounajjed. Part III: Non-neoplastic diseases of the stomach: 6. Common types of gastritis / Michael Torbenson
    7. Special forms of gastritis / Saba Yasir
    8. Drugs-induced injury, infections, vascular, congenital, and miscellaneous disorders / Vishal S. Chandan. Part IV: Non-neoplastic diseases of the duodenum: 9. Malabsorption and malnutrition disorders / Tsung-Teh Wu
    10. Other inflammatory disorders of duodenum / Tsung-Teh Wu
    11. Infections disorders of the duodenum and small bowel / Audrey N. Schuetz
    12. Drug-induced injury, polyps, congenital, and miscellaneous disorders / Vishal S. Chandan and Tsung-Teh Wu. Part V: Non-neoplastic diseases of the jejunum and ileum: 13. Inflammatory bowel disease / Lizhi Zhang
    14. Drug-induced injury, vascular, congenital, and miscellaneous disorders / Lizhi Zhang. Part VI: Non-neoplastic diseases of the colon: 15. Inflammatory bowel disease / Vishal S. Chandan and Tsung-Teh Wu
    16. Non-inflammatory bowel disease colitis / Murli Krishna
    17. Infections disorders of the colon / Bobbi S. Pritt
    18. Drug-induced injury, vascular, congenital, motility, polyps, and miscellaneous disorders / Vishal S. Chandan. Part VII: Non-neoplastic diseases of the appendix and anus: 19. Non-neoplastic diseases of appendix / Samar Said
    20. Non-neoplastic diseases of anus / Sejal Subhash Shah.
    Digital Access Springer 2019
  • Book
    Arnold Goldberg, editor.
    Print 1990
  • Article
    Parry DG, Taylor KW.
    Biochem J. 1978 Mar 15;170(3):523-7.
    1. Rabbit islets of Langerhans were disrupted by ultrasonic methods and the sonicated preparations were used to study proinsulin biosynthesis. 2. When [3h]leucine is incubated in such preparations, incorporation takes place into proinsulin, as evidenced by characterization on polyacrylamide gels, and by the conversion of this labelled material into insulin, by using trypsin. 3. The labelled proinsulin may also be purified by antiinsulin antibody bound to Sepharose. 4. With the broken-cell preparation it was shown that incorporation of leucine is accelerated by increasing the glucose content of the medium from 2mM to 16mM. However, 16mM-galactose or -sucrose did not stimulate incorporation significantly from basal values. This effect of glucose was abolished by cycloheximide. 5. The significance of these findings in relation to the mechanism of glucose stimulation of proinsulin biosynthesis is discussed.
    Digital Access Access Options