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  • Book
    Richard S. Snell.
    Summary: For medical, dental, allied health, and nursing programs, this book guides students through the fundamentals of human anatomy, explaining the how and why behind each structure, and offering readers the hands-on guidance they need to make sound clinical choices. Organized by body region from surface to deep structures, this new edition features: Updated design and layout allowing for a shorter, more focused text. Basic Clinical Anatomy sections with essential information on gross anatomic structures of clinical significance. Chapter Objectives that focus students on material most important to their preparedness for the patient encounter. Clinical Notes highlighting the clinical importance of anatomical information. Clinical Notes highlighting the clinical importance of anatomical information and much much more!

    Contents:
    The thorax. Part I, The thoracic wall
    The thorax. Part II, The thoracic cavity
    The abdomen. Part I, The abdominal wall
    The abdomen. Part II, The abdominal cavity
    The pelvis. Part I, The pelvic walls
    The pelvis. Part II, The pelvic cavity
    The perineum
    The upper limb
    The lower limb
    The head and neck
    The back.
    Digital Access Ovid 2012
  • Article
    Bowden DH, Adamson IY.
    Lab Invest. 1978 Apr;38(4):422-9.
    The adaptive capacity of the lung to increase the output of alveolar macrophages is vital to its handling of foreign material, particularly in an overload situation. In previous papers we demonstrated the role of pulmonary intersititial cells in providing a pool of precursor cells that may be available for this purpose. We now extend these observations to a study of pulmonary cytodynamics in mice subjected to a single large load (4 mg.) of carbon delivered by endotracheal tube. The output of free macrophages over a period of 14 days was correlated with DNA synthesis of pulmonary cells as measured by their uptake of 3H-thymidine. The initial increase of macrophagic output, 5 times in 12 hours and 10 times in 24 hours occurred before any increase in mitotic activity or cellularity was demonstrated in the pulmonary interstitium. The high level of macrophagic cell output which was maintained over the next week was accompanied by increased thymidine uptake in the lung. Increased mitotic activity was confined to the interstitial cell population; no mitoses were observed in free macrophages. The results indicate a biphasic macrophagic response to inhaled particulates, an early phase apparently unrelated to a local cellular response and a later phase of interstitial cell proliferation which appears to be concerned with the maintenance of the high output of macrophages.
    Digital Access Access Options