Today's Hours: 8:00am - 10:00pm

Search

Did You Mean:

Search Results

  • Book
    Giampiero Campanelli, editor.
    Summary: This book is a comprehensive guide to the surgical repair of inguinal and abdominal wall hernias that not only describes all potential approaches, but also places them in the context of the anatomy of the region, the pathology, and the advances in scientific knowledge over the past decade. It documents in detail the individual techniques applicable in each region (inguinal, femoral, and ventral), highlighting tips and tricks and focusing on indications, potential complications, and outcomes. In addition it presents cases of incisional hernia and examines less frequent and rare cases and complex situations. Written for surgeons from around the globe, it includes procedures used in wealthy, developed countries and those without mesh more commonly employed in developing countries. With a format designed to facilitate use in daily practice, it is invaluable for residents seeking step-by-step guidance on procedures ranging from repair of simple inguinal hernias to complex reconstruction; for general surgeons who frequently perform hernia repairs; and for hernia specialists aiming to achieve optimal results. It also appeals to researchers with an interest in the scientific background to hernia surgery.
    Digital Access Springer 2018
  • Article
    McAdam KP, Ryan JL.
    J Immunol. 1978 Jan;120(1):249-53.
    C57BL/10/CR mice do not respond to the lipid A moiety of LPS. This defect was demonstrated in vivo by a decreased production of the acute phase serum amyloid protein SAA. In addition, the defect was demonstrated in vitro by using cultures of spleen cells and peritoneal cells. No mitogenesis of spleen cells or enhanced glucose utilization by either spleen cells or peritoneal cells was observed when the cells were stimulated by lipid A or phenol-extracted Escherichia coli K235 LPS. The response of these mice to PHA, Con A, Poly I:C, 8BrcGMP, and butanol extraced E. coli K235 LPS was normal. Thus, the inability of lipid A to stimulate B lymphocyte mitogenesis and activate peritoneal cells in vitro may correlate with its inability to induce the acute phase SAA in vivo. B10/CR mice represent another strain, similar to the C3H/HeJ, which is nonresponsive to lipid A and should be useful in eludcidating the mechanism by which bacterial LPS activates cells.
    Digital Access Access Options