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  • Book
    edited by Brian Hainline, Robert A. Stern.
    Contents:
    Section I: Introduction. Sports and the human brain: an evolutionary perspective
    Sports neurology as a multidisciplinary field
    Section II: Traumatic brain injury and concussion. Mild traumatic brain injury and concussion: terminology and classification
    Catastrophic neurologic injuries in sport
    Concussive and subconcussive brain trauma: the complexity of impact biomechanics and injury
    risk in contact sport
    Concussion: pathophysiology and clinical translation
    Epidemiology of sports concussion in the United States
    Sideline assessment of concussion
    Emergency department evaluation of the concussed athlete
    Office-based concussion evaluation, diagnosis, and management: adult
    Office-based concussion evaluation, diagnosis, and management: pediatric
    The relationship of migraine and other headache disorders to concussion
    Sleep disorders and concussion
    Vestibular dysfunction and concussion
    Neuro-ophthalmologic disorders following concussion
    Neuropsychiatry of sport-related concussion
    Postconcussion syndrome
    The role of neuropsychologists in concussion evaluation and management
    Return to play following sports-related concussion
    Return to learn
    Neuroimaging of brain trauma in sports
    Blood and cerebrospinal fluid biomarkers
    Measuring head impacts: accelerometers and other sensors
    Subconcussive trauma
    The evolving landscape of policies, rules, and law in sport-related concussion
    The modern landscape of sport-related concussion research: key achievements and future directions
    Section III: Neurodegeneration and other long-term consequences of concussion and
    repetitive head impacts. Chronic traumatic encephalopathy: clinical presentation and in vivo diagnosis
    The neuropathology of chronic traumatic encephalopathy
    Chronic traumatic encephalopathy: neuroimaging biomarkers
    Chronic traumatic encephalopathy: fluid biomarkers
    Section IV: Cervical spinal cord injuries. Biomechanics and common mechanisms of injury of the cervical spine
    Cervical spine trauma evaluation
    Acute management of cervical spine trauma
    Cervical spine trauma: prevention strategies
    Classification in para sport for athletes following cervical spine trauma
    Section V: Peripheral nerve injuries. Peripheral nerve injury in sport: an overview
    Neuromuscular adaptations in shoulder function and dysfunction
    Common peripheral nerve injuries in sport: diagnosis and management
    Section VI: Pain. Sport-related injury and pain classification
    Pain management in sport: therapeutic injections
    Section VII: Special neurologic considerations in sport. Athletes with neurologic disease
    Neurologic benefits of sports and exercise
    Future directions.
    Digital Access ScienceDirect 2018
  • Article
    Melton T, Hartman PE, Stratis JP, Lee TL, Davis AT.
    J Bacteriol. 1978 Feb;133(2):708-16.
    Patterns of chemotaxis by Salmonella typhimurium strain LT-2 to l-amino acids and to several sugars were quantitated by the Adler capillary procedure. Competition experiments indicated that LT-2 possesses three predominant receptors, or interacting sets of receptors, for amino acids. These were termed the aspartate, serine, and alanine classes, respectively. Studies with strains carrying point and deletion mutations affecting components of the phosphoenolpyruvate: glycose phosphotransferase system (PTS) made unlikely a role in primary reception of d-glucose by the three soluble PTS components, namely HPr, enzyme I, and factor III. A ptsG mutant defective in membrane-bound enzyme IIB' of the high-affinity glucose transport system was shown to exhibit normal chemotaxis providing pleiotropic effects of the mutation were eliminated by its genotypic combination with other pts mutations or, phenotypically, by addition of cyclic AMP and substrate. A correlation was demonstrated between chemotaxis to glucose and activity of the low-affinity glucose transport complex, membrane-bound enzymes IIB:IIA, and an enzyme IIB:IIA mutant was shown to have a preponderant defect in chemotaxis to glucose and mannose. Of four systems capable of galactose transport, only the beta-methylgalactoside transport system was implicated in chemotaxis to galactose. Some properties of a mutant possibly defective in processing of signals for chemotaxis to sugars is described.
    Digital Access Access Options