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  • Book
    Vladimír Beneš, Ondřej Bradáč, editors.
    Contents:
    Introduction
    Motivation to this study
    AVM definition and angioarchitecture
    Feeding arteries
    Associated aneurysms
    Nidus component
    Draining veins
    Genetics of arteriovenous malformations
    Epidemiology of AVM
    Physiology of arteriovenous malformations
    Arterial side
    Steal phenomenon
    Normal perfusion pressure breakthrough
    Venous side
    Occlusive hyperemia
    Compartmental model of AVM nidus
    AVM presentation
    Hemorrhagic presentation
    Morbidity and mortality of AVM related intracranial haemorrhage
    Epilepsy presentation
    Focal neurologic deficit (FND)
    Natural course
    AVM grading
    Surgical AVM grading
    Radiosurgical AVM grading
    Imaging
    Catheter angiography
    Magnetic resonance imaging
    Computed tomography
    Treatment for brain AVMs
    Literature series
    Surgery
    Endovascular embolization
    Stereotactic radiosurgery
    Combined treatment
    ARUBA study comments.
    Digital Access Springer 2017
  • Article
    Milch H, Czirók E, Madár J, Semjén G.
    Acta Microbiol Acad Sci Hung. 1977;24(2):127-37.
    Escherichia coli O78: K80 strains isolated from an outbreak of meningitis, sepsis and enteritis in infants, were compared with O78: K80 strains from sporadic cases of enteritis, healthy carriers and animals. The strains were uniform in antigenic structure and phage pattern but differed in colicinogenicity. The epidemic strains and calf-pathogenic cultures produced colicin V, the remaining isolates were characterized by other types of colicin or were not colicinogenic. Col V+ strains multiplied in the mouse peritoneal cavity more readily and killed the animals at significantly lower doses than did col V- strains. One half of antibiotic resistant O78: K80 strains carried R factor. The spread of R factor could be followed by phage restriction experiments.
    Digital Access Access Options