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  • Book
    David Walk, editor.
    Summary: This concise and practical book provides an overview of neuromuscular disorders in clinical practice. Diagnostic approaches are covered along with patient management strategies in an easy-to-understand and introductory framework. Filling a significant gap in the literature, this resource delivers essential information for neurology trainees and practitioners, as well as anyone interested in learning the fundamentals of this important medical subspecialty.

    Contents:
    Chapter 1. Introduction
    Chapter 2. Diseases of Muscle
    Chapter 3. Disorders of Neuromuscular Transmission
    Chapter 4. Motor Neuron Disorders
    Chapter 5. Diseases of Nerve
    Chapter 6. The Diagnostic Approach to the Hypotonic and Weak Infant
    Chapter 7. Rehabilitation Therapists' Role in Management of Neuromuscular Disease
    Chapter 8. Electrodiagnostic Testing
    Chapter 9. Ventilatory Management in Neuromuscular Disease
    Chapter 10. Genetic Testing in Neuromuscular Disease.
    Digital Access Springer 2018
  • Article
    Colman RW, Wong PY.
    Thromb Haemost. 1977 Dec 15;38(4):751-75.
    Abnormalities of Hageman factor dependent pathways have been described in a wide variety of human disease states. Congenital deficiencies of factor XII (Hageman trait) prekallikrein (Fletcher trait) and high molecular weight kininogen (Williams, Fitzgerald and Flaujeac traits) although resulting in profound in vitro changes, do not cause in vivo difficulties. In contrast, deficiency of C1 esterase inhibitor (hereditary angioedema) results in significant morbidity and mortality. Acquired diseases may exhibit decreased synthesis of these three proteins in cirrhosis and dengue fever. In vivo activation of factor XII initiated pathways occur in septic shock, disseminated or localized intravascular coagulation, typhoid fever, polycythemia vera, hyperbetalipoproteinemia, coronary artery disease, nephrotic syndrome, transfusion reactions, hemodialysis and extracorporeal bypass. Activation of both the intrinsic system and tissue mediators contribute to the vasomotor phenomena in carcinoid syndrome and postgastrectomy dumping. Roles for factor XII, prekallikrein and kininogen have been suggested in gouty arthritis, allergic disorders and cystic fibrosis but the evidence is not yet convincing in these disorders.
    Digital Access Access Options