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- BookChristos P. Panteliadis, editor.Summary: This third edition systematically reviews recent developments in the diagnosis and evidence-based treatment of cerebral palsy, a consequence of foetal and early infant brain damage resulting in lifelong disabilities with a range of clinical characteristics. The first part discusses the definition, aetiology, classification, imaging and neuropathology, while the second focuses on the management of the individual challenges that children with cerebral palsy face, such as spasticity, dyskinesia, feeding problems and scoliosis. Based on the diverse characteristics of cerebral palsy, children require care from various specialists, including neuro-paediatricians, orthopaedists, psychologists, epidemiologists, physiotherapists and occupational therapists. This work was written by an international team of such specialists, providing a comprehensive mix of perspectives and expertise.
Contents:
Cerebral palsy: a historical review
Definition
Epidemiology
Neuropathology
Aetiological factors
Intrauterine infection
Magnesium sulphate for prevention
Early markers
Clinical characteristics
Early diagnosis
Brain Imaging
Ultrasonography
Positrons-Emission-Tomography
Physiotherapy
Occupational therapy
Pathophysiology of muscle
Orthpaedic management
Early developmental intervention
Hip luxation
Scoliosis
Bone status
Oral medication
Management with Botulinum Neurotoxin A
Intrathecal Baclofen
Rhizotomy
Comorbidities.- Epilepsies
Visual impairment
Gastrointestinal problems
Long-term prognosis
Quality of life
Psychosocial problems.Digital Access Springer 2018 - BookJean Ziegler ; aus dem französischen übersetzt von Friedrich Griese und Thorsten Schmidt.Print c1990
- ArticleMoser GC, Meiss HK.Somatic Cell Genet. 1977 Jul;3(4):449-56.Interphase cells stained with quinacrine dihydrochloride show distinctive fluorescent nuclear patterns according to their position in the cell cycle. These patterns were used to determine whether temperature-sensitive growth mutants of the Syrian hamster cell line BHK-21 are blocked at specific stages of the cell cycle. These cytological studies confirmed the previous conclusion that ts Af8 cell line derived from BHK cells is a GI cell-cycle mutant. The method promises to be of use as an initial probe in screening for cell-cycle mutants.