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  • Book
    editors, Craig S. Kitchens, Craig M. Kessler, Barbara A. Konkle, David A. Garcia.
    Summary: "With authoritative coverage of rare and common hemostatic disorders, Consultative Hemostasis and Thrombosis, 4th Edition, keeps you both up to date with all that's new in this fast-moving field as well as reviewing background and development and citing pertinent classical literature. Broad differential diagnoses are provided, underscoring the editors' position that correct treatment begins with correct diagnosis"--Publisher's description.

    Contents:
    Part 1: Hemorrhagic processes. Consultative process
    An approach to the bleeding patient: correlation of clinical symptoms and signs with laboratory testing
    Hemophilia A and B
    Less common congenital disorders of hemostasis
    Acquired coagulation disorders due to inhibitors
    von Willebrand disease
    General aspects of thrombocytopenia, platelet transfusions, and thrombopoietic growth factors
    Primary immune thrombocytopenia
    Congenital and acquired disorders of platelet function and number
    Purpura and other hematovascular disorders
    HHT
    Disseminated intravascular coagulation
    The cross-talk of inflammation and coagulation in infectious disease and their roles in disseminated intravascular coagulation
    Part 2: Thrombotic processes. Thrombophilia: clinical and laboratory assessment and management
    Pediatric aspects of thrombophilia
    Deep vein thrombosis, pulmonary embolism and primary pulmonary hypertension
    Venous thromboses at unusual sites
    The post thrombotic syndrome
    Thrombocytosis: essential thrombocytemia and reactive causes
    Antiphospholipid syndrome
    Antiplatelet treatment in cardiovascular medicine
    Non-arteriosclerotic arterial occlusive disease
    Thrombosis and cancer
    Thrombotic thrombocytopenic purpura
    Hemostatic disorders of complement activation (PNH and aHUS)
    Heparin-induced thrombocytopenia
    Part 3: Therapeutic agents. Antithrombotic agents
    Blood component and pharmacological agents
    Therapeutic apheresis: applications for hemorrhagic & thrombotic disorders
    Vena caval filters
    Part 4: Issues specific to women. Thrombotic risk of contraceptives and other hormonal therapies
    Bleeding and the management of hemorrhagic disorders in pregnancy
    Thrombophilia in pregnancy
    Part 5: Special issues. Surgery and hemostasis
    Anticoagulation in the perioperative period
    Understanding and managing the coagulopathy of liver disease
    Outpatient anticoagulant therapy
    Hematologic interventions for acute central nervous system disease
    Pending: atrial septal abnormalities and cryptogenic stroke
    Hemorrhage control and thrombosis following severe injury
    Hemostatic aspects of sickle cell disease
    Digital Access ClinicalKey 2019
  • Article
    Schmidt P, Zazgornik J, Kopsa H, Balcke P, Pils P, Bayer PM.
    Wien Klin Wochenschr. 1977 Dec 23;89(24):819-21.
    Regular 4-weekly follow-up controls of serum lactate dehydrogenase activity in 23 renal transplant recipients revealed a constant rise in serum LDH activity during the early postoperative months. During the first post-transplant month serum LDH activity increased from 150.0 +/- 48.2 mE/ml to 195.5 +/- 84.8 mE/ml, serum enzyme activity being highest (329.1 +/- 143.2 mE/ml) 6 months after surgery. Since serum creatinine levels remained relatively constant, it seems unlikely that renal rejection played a major pathogenic role in the production of increased LDH activity. Since the pattern of lactate dehydrogenase isoenzymes was ithin normal limits, the pathogenesis of increased LDH serum activity following renal transplantation is not yet clear. Possible causes such as liver damage due to hepatitis B, macrocytosis induced by immunosuppressive therapy and myopathy to steroids are discussed.
    Digital Access Access Options