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  • Book
    Byung-Boong Lee, Stanley G. Rockson, John Bergan, editors.
    Summary: The second edition of this book serves as a central source of theoretical and practical knowledge to optimize the evaluation and treatment of patients with lymphedema. The book covers all aspects of the disease from anatomical and histological features to diagnosis as well as physical/medical and surgical management of the disease. Updated from the first edition to reflect the substantial progress in diagnostics, medical care and surgical intervention for this patient population, this volume has been reorganized to meet today's practice requirements. It addresses the challenges faced by clinicians in the management of chronic lymphedema enabling them to meet the medical needs of this large patient community. Edited by world leaders in Vascular Medicine and Surgery, this comprehensive volume provides clear, concise background and recommendations in an easy-to-use format. It is a valuable reference tool for clinical practitioners (physicians/nurse practitioners/technicians) who wish to deliver state-of-the-art health care to their patients with lymphatic and venous disorders.

    Contents:
    Section I
    Introduction
    1. General Considerations
    2. Etiology and Classification of Lymphatic Disorders
    3. Hereditary and Familial Lymphedemas.-Section II
    Embryology, Anatomy, & Histology
    4. Embryology of the Lymphatic System and Lymphangiogenesis
    5. Anatomy of the Lymphatic System and Its Structural Disorders in Lymphoedema
    Section III
    Physiology, Pathophysiology and Lymphodynamic
    6. General Overview
    7. Lymphodynamics
    8. Physiology- Lymph Flow
    9. Biomechanics of the Lymphatic Circulation
    10. Pathology & Histochemistry
    11. Lymph Formation and Composition
    Section IV
    Clinical Diagnosis
    12. General Overview
    13. Lymphedema Epidemiology.-14. Clinical Staging
    15. Combined Clinical and Laboratory (Lymphoscintigraphic) Staging
    16. Early Diagnosis in the Latent Phase
    17. Cutaneous Manifestations of Edema
    18. The Diagnosis of Edema and Its Pathogenesis
    19. Differential Diagnosis: Venous Edema
    20. Differential Diagnosis: Lipedema.-Section V
    Laboratory/Imaging Diagnosis
    21. General Guidelines
    22. Radionuclide Lymphoscintigraphy
    23. Duplex ultrasonography
    24. Oil Contrast Lymphangiography
    25. Microscopic Lymphoangiography
    26. Near Infrared Fluorescent Lymphography
    27. MR Lymphangiography
    28. Combined role of the lymphoscintigraphy, Xray Computed Tomography, Magnetic Resonance Imaging, and Positron Emission Tomography in the Management of Lymphedematous Disease
    29. Alternative Assessment & Measurement Tools
    Section VI
    Physical and Medical Management
    30. General Overview
    31. Complete Decongestive Physiotherapy
    32. Decongestive Lymphatic Therapy
    33. Compression Therapy
    34. Intermittent Pneumatic Compression Therapy
    35. Other contemporary Treatment Modalities
    36. Medical Treatment Options
    37. Diagnosis and Managment of Infection in Lymphedema
    38. The Prospect for Genetic and Growth Factor Therapy
    39. Adherence and Quality of Life
    40. Lymphedema within the Healthcare System
    Section VII
    Practical issues in the Physiotherapeutic Approach to Lymphedema
    41. Lower Limb Lymphedema
    42. Upper Limb Lymphedema
    43. Head, Face and Neck Lymphedema
    44. Genital Lymphedema
    Section VIII
    Surgical Treatment
    Reconstructive Surgery
    45. General Overview
    46. Principles of Patient Selection for Surgical Management of Lymphedema
    47. Lymphatic-Venous Derivative and Reconstructive Microsurgery
    48. Lymphatic-Lymphatic Reconstructive Microsurgery
    49. Lymph Node-Venous Microvascular Reconstructive Surgery: Filariasis Lymphedema
    50. Vascularized Lymph Node Transfer for the Treatment of Lymphedema
    51. A Combined Microsurgical Reconstruction Approach for Lymphedema
    52. Current Dilemmas and Controversy in Reconstructive Surgery for Lymphedema
    53. Future Prospects in Lymphatic Reconstructive Surgery
    Section IX
    Surgical treatment: Excisional and Debulking Techniques
    54. General Principles
    55. Contemporary Indications and Controversies in Excisional Surgery
    56. From Lymph to Fat
    Liposuction as a Treatment for Complete Reduction of Lymphedema
    57. Surgical Management of Lipedema
    Section X
    Congenital Vascular Malformation with Lymphatic Involvement
    58. General Overview
    59. Primary lymphedema and Lymphatic Malformation
    60. Molecular Genetics of Lymphatic and Complex Vascular Malformations
    61. Syndromic Lymphedema and Complex Vascular Malformations with Lymphatic Involvement
    62. An Atlas of Neonatal and Infantile Lymphedema
    Section XII
    Management of Chyle Reflux and Effusions.-63. Pathophysiology and Medical Management of Chylous Disorders
    64. Surgical Management of Chylous Reflux and Effusions
    65. Endovascular Catheter-based Management of Chylous Effusions.-Section XII
    Lymphatic Filariasis
    66. Epidemiology
    67. Etiology & Pathophysiology
    68. Clinical Overview- Diagnosis and Management
    Section XIII
    Oncology and Lymphedema
    69. Breast Cancer.-70. Lower Extremity Cancers
    71. Radiation Considerations
    Section
    XIV
    Phlebolymphedema
    72. Diagnosis and Management of Primary Phlebolymphedema
    73. Diagnosis and Management of Secondary Phlebolymphedema
    74. Management of Phlebolymphedema Ulcer.
    Digital Access Springer 2018
  • Article
    Bereziat G, Pepin D, Wolf C, Polonovski J.
    Pathol Biol (Paris). 1977 Oct;25(8):559-64.
    The level of the four major bile acids was measured in the plasma using gas liquid chromatography. The enhancement of the level of the primary bile acids was well known during bile duct obstruction. In the case of hepatic diseases, the chenodesoxycholiccholic acid concentration ratio (CDC/C) was equal or lower than unity when cholestasis occurred rather than hepatic deficiency. When hepatic deficiency developed, the ratio CDC/C was higher than unity. Plasma bile acid fractionnation exhibits also a prognostic value. When the ratio CDC/C is very high, it is significant of progressive hepatic deficiency. A high level of lithocholic acid is also a sign of unfavourable prognosis.
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