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  • Book
    Keyvan Moharamzadeh.
    Contents:
    1. Aggressive periodontitis
    2. Amelogenesis imperfecta
    3. Apical periodontitis
    4. Bisphosphonates and medication-related osteonecrosis of the jaw
    5. Caries
    6. Chronic periodontitis
    7. Cleft lip and palate
    8. Combination syndrome
    9. Complete edentulism
    10. Deep overbite
    11. Dens invaginatus
    12. Dentine hypersensitivity
    13. Dentinogenesis imperfecta and dentine dysplasia
    14. Dentoalveolar trauma
    15. Discolouration
    16. Ectodermal dysplasia
    17. External root resorption
    18. Failed restoration and compromised tooth
    19. Fluorosis
    20. Fractured endodontic instrument
    21. Fractured or failed post
    22. Gingival enlargement and gingivitis
    23. Gingival recession
    24. Halitosis
    25. Hypodontia
    26. Internal root resorption
    27. Irrigation accidents
    28. Low maxillary sinus floor
    29. Microdontia
    30. Necrotising periodontal diseases
    31. Occlual issues and occlusion
    32. Oncology-related defects in mandible
    33. Oncology-related defects in maxillae
    34. Open apex (immature non-vital teeth)
    35. Open bite
    36. Osteoradionecrosis
    37. Partial edentulism- implant treatment
    38. Partial edentulism- partial denture treatment
    39. Perforation
    40. Peri-implant disease
    41. Periodontal abscess
    42. Periodontic-endodontic lesion
    43. Periodontitis as a manifestation of systemic disease
    44. Radiotherapy side effects
    45. Ridge defects (generalised)
    46. Ridge defects (localised)
    47. Shortened dental arch (SDA)
    48. Supernumerary, fusion and gemination
    49. Temporomandibular disorders
    50. Tooth wear
    Index.
    Digital Access Wiley 2018
  • Book
    edited by Diane E. Marting.
    Print 1990
  • Article
    Schiefer HG, Gerhardt U, Brunner H.
    Zentralbl Bakteriol Orig A. 1977 Oct;239(2):262-9.
    A phosphoglycolipid, presumably identical to glyceryl-phosphoryldiglycosyl diglyceride, is the main component of the membrane glycolipids of Mycoplasma mycoides subsp. capri. It is immunologically active. Anti-phosphoglycolipid antibodies were induced in rabbits by intravenous injection of the flocculated complexes of methylated bovine serum albumin with a mixture of the phosphoglycolipid and the auxiliary lipids, phosphatidyl-choline and cholesterol. The specificities of the antibodies directed against the phosphoglycolipid, are due to both the phosphate and carbohydrate moieties of the lipid molecule. Anti-phosphoglycolipid antibodies were detected in the sera of rabbits intravenously immunized with intact M. mycoides subsp. capri. The intravenous method of immunization was chosen in order to select for a response to surface antigenic determinants. Anti-phosphoglycolipid antibodies specifically reacted with intact organisms and isolated membranes of M. mycoides subsp. capri, as shown by complement fixation and agglutination tests. The antigenic determinants of the phosphoglycolipid are mainly located on the outer membrane surface. It is concluded that the antigenic determinants of the phosphoglycolipid in intact M. mycoides subsp. capri significantly contribute to the surface architecture of mycoplasma membranes.
    Digital Access Access Options