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  • Book
    editors; M.F. Chernov, Y Muragaki, S. Kesari, I.E. McCutcheon.
    Contents:
    Pathology and genetics of gliomas / Komori T., Muragaki Y., Chernov M.F.
    Fractionated radiotherapy of intracranial gliomas / Ghia A.J.
    Stereotactic radiosurgery in the multimodality management of residual or recurrent glioblastoma multiforme / Niranjan A., Monaco E.A. III, Kano H., Flickinger J.C., Lunsford L.D.
    Stereotactic radiosurgery of intracranial low-grade gliomas / Ganz J.C.
    Brachytherapy of intracranial gliomas / Nachbichler S.B., Kreth F.-W.
    Irradiation of intracranial gliomas in children / Kortmann R.-D., Seidel C., Müller K., Hirsch F.W.
    Role of radiosensitizers in radiation treatment of gliomas / Cohen R.J., Mehta M.P.
    Chemotherapy of high-grade astrocytomas in adults / Puduvalli V.K., Hoang N.
    Chemotherapy of diffuse astrocytoma (WHO grade II) in adults / Narita Y.
    Chemotherapy of oligodendrogliomas / Drappatz J., Lieberman F.
    Chemotherapy of intracranial gliomas in children / Terashima K.
    Perspectives of personalized chemotherapy of gliomas based on molecular tumor profiling / Soffietti R., Franchino F., Magistrello M., Pellerino A., Rud̉ R.
    Antiangiogenic therapy of high-grade gliomas / Jo J., Wen P.Y.
    Search for more effective chemotherapeutic regimens for glioma / Physical and Psychological Rehabilitation of Patients with Intracranial Glioma / Andrejeva J., Volkova O.V.
    Palliative and supportive care of patients with intracranial glioma / Pace A., Villani V.
    Digital Access Karger 2018
  • Article
    Krafft T, Peitrequin R, Frei PC.
    Schweiz Med Wochenschr. 1977 Dec 03;107(48):1759-61.
    Circulating immune complexes were measured by a radioimmunoassay specific for HB complexes in different clinical situations related to hepatitis B virus infection. The leukocyte migration inhibition test (LMIT) was performed simultaneously. Comparison with the clinical situation suggests that immune complexes are not responsible for the lesions, but that these lesions might be produced by the immunity demonstrated by the LMIT. A deficiency of this immunity is responsible for the persistence of infection. However, in chronic active hepatitis, LMIT is as deficient as in carriers and in this form of hepatitis the frequency and quantity of complexes are the highest: it is thus possible that in this case the lesions are nevertheless produced by the complexes.
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