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- BookGeorge J. Brewer.Summary: Environmental Causes and Prevention Measures for Alzheimer's Disease examines the increased incidence of the disease in developed countries and aims to educate neuroscientists, medical practitioners and other educated individuals on new insights into environmental causation, primarily metals. This book looks into the web of evidence around the hypothesis of copper toxicity and the additional role that a high fat diet plays in disease progression and cognition loss. The data and its implications are discussed, along with potential prevention measures. This book will generate excitement and interest among neuroscientists, medical practitioners and other biomedical researchers.
Contents:
A little background on dementia and Alzheimer's disease
Interesting and important historical and demographic facts about the epidemic of Alzheimer's disease in developed countries pointing to environmental intoxicants causing the epidemic
Candidate environmental factors for the Alzheimer's epidemic part 1: The metals-aluminum, lead, mercury, zinc, iron, and copper
Candidate environmental factors for the Alzheimer's epidemic part 2: diet and other lifestyle factors
Identification of copper-2 and cooper in general, as major environmental intoxicants in the Alzheimer's disease epidemic: the copper hypothesis
Background on copper, including why copper-2 is so specificially neurotoxic
Inorganic copper, or copper-2, ingestion as a major causal factor for the Alzheimer's disease epidemic
the web of evidence
Increased copper absorption resulting from dietary changes in developed countries as another causal factor in the Alzheimer's disease epidemic
The copper hypothesis fits nicely with known risk factors and theories of Alzheimer's disease causation
Prevention measures action items: two simple steps to eliminate ingestion of copper-2, and dietary changes to reduce copper absorption
Failures: what the government has not done to ensure healthy drinking water and nontoxic multimineral pills
Treatment of Alzheimer's disease
Sumary and conclusions.Digital Access ScienceDirect 2018 - Bookproject team, Mark E. Monaco ... [et al.].Contents:
V. 1. Data summaries
V. 2. Species life history summariesDigital Access Full text via HathiTrust, [1990]- - ArticleBerdicevsky I, Grossowicz N.J Gen Microbiol. 1977 Oct;102(2):299-304.The polyene antibiotic, amphotericin, inhibited phosphate uptake in Candida albicans more strongly than it inhibited growth. Cultures grown from an inoculum of young (2 h) cells were more affected than those inoculated with old (24 h) cells. Thus, the polyene displays a double effect on C. albicans (and presumably on other eukaryotic cells): it interferes with membrane sterols and also inhibits synthesis of a factor (or factors) during growth. Whether this factor(s) interferes with the uptake of the polyene antibiotic or neutralizes its effect by reacting with it remains unsolved.