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- Book[edited by] Eric D. Hsi.Contents:
Non-neoplastic disorders. Red blood cell/hemoglobin disorders
Coagulation
Non-neoplastic morphologic abnormalities of white blood cells and macrophages
Reactive lymph nodes and castleman disease
Bone marrow failure syndromes
Benign causes of bone marrow abnormalities including infections, storage diseases, systemic disorders, and stromal changes
Lymphomas
Small B-cell lymphomas
Diffuse aggressive B-cell lymphomas
Peripheral T-cell lymphomas
Immunodeficiency-related lymphoproliferative disorders
Hodgkin lymphoma
Leukemias, chronic myeloproliferative neoplasms, and myelodysplasia
B-cell leukemias of mature lymphocytes
Mature T-cell and natural killer cell leukemias
Acute myeloid leukemia
Precursor lymphoid neoplasms
Acute undifferentiated leukemia and mixed-phenotype acute leukemias
Myeloproliferative and "overlap" myelodysplastic/myeloproliferative neoplasms
Myelodysplastic syndromes
Disorders of histiocytes, mast cells, plasma cells, spleen, and ancillary techniques
Disorders of histiocytes
Mastocytosis
Multiple myeloma and other plasma cell neoplasms
Disorders of the spleen
Flow cytometric principles in hematopathology
Molecular diagnosis in hematopathologyDigital Access ClinicalKey 2018 - ArticleDeLeys RJ, Juni E.Antimicrob Agents Chemother. 1977 Nov;12(5):573-6.Growth of exponential-phase liquid cultures of Moraxella osloensis was inhibited by 0.5 U of penicillin G per ml. For this organism, low concentrations of penicillin acted primarily in a bacteriostatic rather than in a bactericidal manner. At higher concentrations of penicillin some killing did take place, but the rate of killing was rather slow and appeared to be independent of penicillin concentration. Microscopic observation of cells from penicillin-treated cultures showed little or no cellular swelling or lysis. The total cell count did not decrease significantly during 6 h of incubation in 5,000 U of penicillin per ml. The rates of respiration, nucleic acid synthesis, and protein synthesis were not affected by the presence of penicillin. Attempts to counteract the bactericidal action of high concentrations of penicillin with growth inhibitory concentrations of chloramphenicol were unsuccessful, since chloramphenicol itself was more bactericidal than penicillin for M. osloensis.