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  • Book
    edited by J.P.F. D'Mello, formerly of SAC, University of Edinburgh King's Buildings Campus, Edinburgh, UK.
    Digital Access Ovid 2017
  • Book
    15th Colloquium on Trees in Algebra and Programming, Copenhagen, Denmark, May 15-18 1990, proceedings ; A. Arnold, (ed.).
    Summary: This volume contains the proceedings of the Fifteenth Colloquium on Trees in Algebra and Programming. The papers selected present new research results and cover the following topics: - Logical, algebraic and combinatorial properties of discrete structures (strings, trees, graphs, etc.), including the theory of formal languages considered as that of sets of discrete structures and the theory of rewriting systems over these objects. - Application of discrete structures in computer science, including syntax and semantics of programming languages, operational semantics, logic programming, algorithms and data structures, complexity of algorithms and implementation aspects, proof techniques for nonnumerical algorithms, formal specifications, and visualization of trees and graphs. Nielsen 9783540525905 20160528
    Digital Access
    Provider
    Version
    SpringerLink
    SpringerLink, c1990
    SpringerLink
    SpringerLink, c1990
  • Article
    Pace CS, Murphy M, Conant S, Lacy PE.
    Am J Physiol. 1977 11;233(5):C165-71.
    Electrophysiological studies of rat islet cells in monolayer culture were undertaken to determine the role of transmembranous ionic fluxes in the inhibitory action of somatostatin on insulin release. In the presence of somatotropin release inhibiting factor (SRIF) (2.5 nM), hyperpolarization occured with or without glucose (16.6 mM) in the medium. SRIF also inhibited the incidence of glucose-induced spike activity. The inhibitory action of SRIF occurred within 5 min and was readily reversible. An increase in extracellular K+ (5-13 mM) or Ca2+ (2.3-4.6 mM) prevented SRIF inhibition of glucose-induced electrical activity. The secretory response of cultured islets to glucose (16.6 mM) was completely inhibited by SRIF (2.5 nM). The presence of high [Ca2+]o or [k+]o enhanced insulin release in the presence of SRIF and glucose. Although phentolamine (5.0 microgram/ml) did not block the inhibition of glucose-induced electrical responses by SRIF, it prevented the inhibitory action of epinephrine (0.2 microgram/ml). It is concluded that the primary action of SRIF is to alter transmembranous cationic fluxes, as manifested by hyperpolarization and a decrease in the incidence of spike activity, which may prevent glucose from eliciting a normal secretory response.
    Digital Access Access Options