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  • Book
    edited by Sam Ghebrehewet, Alex G. Stewart, David Baxter, Paul Shears, David Conrad, and Merav Kliner.
    Summary: A practical guide for practitioners working at all levels in public health and health protection. It is aimed at individuals training in health protection and public health including those with a non-specialist background.

    Contents:
    Section 1. The basics
    Section 2. Infectious disease control case studies and scenarios
    Section 3. Emergency preparedness, resilience and response (EPRR), and business continuity case studies and scenarios
    Section 4. Environmental public health practice case studies and scenarios
    Section 5. Health protection tools
    Section 6. New and emerging health protection issues.
    Digital Access Oxford 2016
  • Article
    DiBona GF.
    Am J Physiol. 1977 Aug;233(2):F73-81.
    The evidence supporting a role for direct neurogenic control of renal tubular sodium reabsorption is reviewed. Electron microscopic and fluorescence histochemical studies have demonstrated adrenergic nerve terminals in direct contact with basement membranes of mammalian (rat, dog, and monkey) renal tubular epithelial cells. Low-level direct or baroreceptor reflex stimulation of renal sympathetic nerves produces an increase in renal tubular sodium reabsorption without alterations in glomerular filtration rate, renal blood flow, or intrarenal distribution of blood flow. Antinatriuresis was prevented by prior treatment of the kidney with guanethidine or phenoxybenzamine. Rat kidney micropuncture studies have localized a site of enhanced tubular sodium reabsorption to the proximal tubule. Possible indirect mediation of the antinatriuresis by other humoral agents known to be released from the kidney on renal nerve stimulation (angiotensin II, prostaglandin) was excluded by experiments with appropriate blocking agents. The possible effects of anesthesia and uncertainties about the completeness of surgical renal denervation and other tubular segmental sites of action are critically analyzed. The clinical implications of this mechanism in pathologic conditions of sodium and water retention are discussed and and a prospectus for future work is presented.
    Digital Access Access Options