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- BookClaudia Stange.Contents:
Carotenoid distribution in nature
Biosynthesis of carotenoids in plants: enzymes and color
Structures and analysis of carotenoid molecules
Carotenoids and photosynthesis
Regulation of carotenoid biosynthesis in photosynthetic organs
Regulation of carotenoid biosynthesis during fruit development
Carotenoid biosynthesis in Daucus carota
Carotenoids in microalgae
Apocarotenoids : a new carotenoid-derived pathway
Plastids and carotenoid accumulation
Evidence of epigenetic mechanisms affecting carotenoids
Manipulation of carotenoid content in plants to improve human health
Modern breeding and biotechnological approaches to enhance carotenoid accumulation in seeds
Carotenoids as a source of antioxidants in the diet
Carotenoids in adipose tissue biology and obesity
Absorption of carotenoids and mechanisms involved in their health-related properties.Digital Access Springer 2016 - ArticleCoronary Drug Project Research Group.N Engl J Med. 1977 05 26;296(21):1185-90.We analyzed data obtained during the Coronary Drug Project to discover the influence of the drugs used on the frequency of gallbladder disease. Of 2680 placebo-treated men who had had myocardial infarction, gallbladder disease developed in 69. Corresponding figures for those given 2.5 mg of estrogen, 5.0 mg of estrogen and 1.8 g of clofibrate per day were 46 of 1061, 47 of 1081 and 42 of 1051, respectively. Each treatment group differed from placebo by over twice the standard error of the difference, life-table analysis yielding P less than 0.05 for each drug-placebo comparison. Forty-five variables, including age, body weight, blood pressure, serum lipids and blood sugar, were evaluated as risk factors. Age significantly correlated with prevalence of known gallbladder disease at entry (r = 0.066, P less than 0.001). No variable yielded a strong and consistent correlation with the incidence of subsequent new gallbladder disease. Gallstone formation is a risk whenever clofibrate or estrogen is prescribed.