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  • Book
    Thomas Böldicke, editor.
    Contents:
    Introduction
    Part I Methods of Structure Prediction
    Chapter 1 In Silico Prediction of Target-Inhibitor Interactions
    Part II Antibodies
    Chapter 2 Antibodies and Selection of Monoclonal Antibodies
    Chapter 3 Selection of Recombinant Human Antibodies
    Chapter 4 Selection of Recombinant Human Antibodies against Toxins and Viruses
    Chapter 5 Recent Advances with ER Targeted Intrabodies
    Chapter 6 Therapeutic Blocking Antibodies against Oncogenic Receptors and Growth Factors
    Chapter 7 Synthetic Cystine-Knot Miniproteins-Valuable Scaffolds for Polypeptide Engineering
    Part III Peptides, Small Molecules and Aptamers
    Chapter 8 Peptides and Peptide Analogs to Inhibit Protein
    Chapter 9 Allosteric Modulators of the Class A G Protein coupled Receptors
    Chapter 10 Phosphatases: Their Roles in Cancer and their Chemical Modulators
    Chapter 11 Selection and Application of Aptamers and Intramers
    Part IV Angiogenesis Inhibitors
    Chapter 12 Inhibitors of Angiogenesis.
    Digital Access Springer 2016
    Access via Advances in experimental medicine and biology ; 2016; 917
    Location
    Version
    Call Number
    Items
    Advances in experimental medicine and biology ; 2016; 917
    2016
  • Article
    Bruni CB, Colantuoni V, Sbordone L, Cortese R, Blasi F.
    J Bacteriol. 1977 Apr;130(1):4-10.
    Escherichia coli K-12 hisT mutants were isolated, and their properties were studied. These mutants are derepressed for the histidine operon, map close to the purF locus at about 49.5 min on the E. coli linkage map, and lack pseudouridylate synthetase activity. The defect in this enzyme leads to the absence of pseudouridines in the anticodon loop of several transfer ribonucleic acid species, as evidenced by the altered elution profile on reversed-phase chromatography and resistance to amino acid analogues. Finally, the hisT mutants studied have a reduced growth rate that appears to be linked to hisT, although it is not known whether it is due to the same mutation. The normal generation time can be restored by supplementing the medium with adenine, uracil, and isoleucine.
    Digital Access Access Options