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  • Book
    Shozo Tobimatsu, Ryusuke Kakigi, editors.
    Contents:
    Part I: Introduction
    1. Principles of Magnetoencephalography
    Part II: Motor System
    2. Basic Function and Clinical Application
    Part III: Somatosensory System
    3. Basic Function
    4. Clinical Applications
    Part IV: Auditory System
    5. Basic Function
    6. Clinical Applications
    Part V: Visual System
    7. Basic Function
    8. Clinical Applications
    Part VI: Epilepsy
    9. Childhood Epilepsy
    10. Adult Epilepsy
    Part VII: Neurological Disorders
    11. Cerebrovascular Diseases
    12. Neurodegenerative Disorders
    Part VIII: Psychiatric Disorders
    13. Autism Spectrum Disorder
    14. Schizophrenia
    15. Bipolar Disorder
    Part IX: Cognition and Brain-machine interface
    16. ECoG Based BCI for BCI-MEG Research
    17. Oscillation and Cross-Frequency Coupling.- .
    Digital Access Springer 2016
  • Book
    by Harold P. Guy.
    Digital Access Full text via HathiTrust, 1970
  • Article
    Fresno M, Jiménez A, Vázquez D.
    Eur J Biochem. 1977 Jan;72(2):323-30.
    The Cephalotaxus alkaloids harringtonine, homoharringtonine and isoharringtonine inhibit protein synthesis in eukaryotic cells. The alkaloids do not inhibit, in model systems, any of the steps of the initiation process but block poly(U)-directed polyphenylalanine synthesis as well as peptide bond formation in the fragment reaction assay, the sparsomycin-induced binding of (C)U-A-C-C-A-[3H]Leu-Ac, and the enzymic and the non-enzymic binding of Phe-tRNA to ribosomes. These results suggest that the Cephalotaxus alkaloids inhibit the elongation phase of translation by preventing substrate binding to the acceptor site on the 60-S ribosome subunit and therefore block aminoacyl-tRNA binding and peptide bond formation. However, the Cephalotaxus alkaloids do not inhibit polypeptide synthesis and peptidyl[3H]puromycin formation in polysomes. Furthermore, these alkaloids strongly inhibit [14C]trichlodermin binding to free ribosomes but hardly affect the interaction of the antibiotic with yeast polysomot interact with polysomes and therefore only inhibit cycles of elongation. This explains the polysome run off that has been observed by some workers in the presence of harringtonine.
    Digital Access Access Options