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  • Video
    Frank Ochberg, MD.
    Summary: Helping children who experience PTSD as a result of a one-time act of violence or trauma can be unsettling for many health professionals, counselors, school personnel or caretakers. This video presents information on the symptoms of PTSD in children and the various stages of treatment for children faced with stress disorder. It gently emphasizes the spiritual aspect of coping strategies and offers sound advice on short-term, long-term and psychopharmacological therapy and the extenuating circumstances of treating PTSD in inner city children. Five noted PTSD specialists who include Frank Ochberg, Carl Bell, Kathleen Nader, Erwin Parson and Angie Panos present the most current information on the study of PTSD in children and its relevance in our communities. This video is an excellent teaching tool for all individuals whose lives are dedicated to the resolution of traumatic experience. Specifically, therapists, social workers, school personnel, health professionals, and all members of community "critical incident" teams would benefit from viewing this presentation. Keywords: PTSD, trauma, child, stress, abuse, violence, violent, children, Counseling, counselling, Social Work, Social Worker, Therapy, Psychotherapy, Psychotherapy.net, Therapist.--Supplied by publisher.
    Digital Access 2011
  • Article
    LaRusso NF, Szczepanik PA, Hofmann AF.
    Gastroenterology. 1977 Jan;72(1):132-40.
    The effect of deoxycholate ingestion, 750 mg per day, on bile acid kinetics, biliary bile acid composition, and biliary lipid secretion was studied in 7 healthy volunteers. Bile acid kinetics were measured by isotope dilution, and hourly outputs of bile acid, cholesterol, and phospholipid were quantitated by a duodenal perfusion technique during a 24-hr period which included three liquid meals and an overnight fast. Biliary bile acid composition was assessed by coupled gas chromatography-mass spectrometry. After deoxycholic acid ingestion, biliary bile acids became composed of predominantly deoxycholyl conjugates, and deoxycholic acid pools increased 4-fold. Both chenodeoxycholic and cholic acid pools decreased, and daily synthesis of each of the primary bile acids was inhibited by 50%. Total bile acid pools did not change in any consistent manner. Daily bile acid secretion increased slightly during deoxycholic acid ingestion, and recycling frequency varied reciprocally with the total bile acid pool both before and during deoxycholic acid treatment. Deoxycholic acid ingestion caused no change in either the daily secretion of cholesterol or lecithin, or the cholesterol saturation of fasting-state bile, which remained unsaturated throughout the study. SGOT levels increased to 4 times the upper limits of normal in 2 of 7 subjects, but these levels promptly returned to normal when deoxycholate feeding was stopped. Serum cholesterol levels decreased in every subject (average 15%) during deoxycholic acid administration. No evidence for a direct role of deoxycholate in the pathogenesis of cholesterol cholelithiasis was obtained in these studies.
    Digital Access Access Options