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  • Book
    edited by Brian C. Gilger.
    Contents:
    Challenges in ocular pharmacokinetics, pharmacodynamics, and toxicology / Brian C. Gilger
    Selection of appropriate animal models in ocular research : ocular anatomy and physiology of common animal models / Brian C. Gilger, Eva Abarca, and Jacklyn H. Salmon
    Challenges and strategies in drug residue measurement (bioanalysis) of ocular tissues / Poonam R. Velagaleti and Michael H. Buonarati
    Chemistry, manufacturing, and control of ophthalmic formulations / Malay Ghosh and Imran Ahmed
    ADME and ocular therapeutics : retina / Cornelis J. Van der Schyf, Samuel D. Crish, Christine Crish, Denise Inman, and Werner J. Geldenhuys
    Compositions, formulation, pharmacology, pharmacokinetics, and toxicity of topical, periocular, and intravitreal ophthalmic drugs / Kishore Cholkar, Aswani Dutt Vadlapudi, Hoang M. Trinh, and Ashim K. Mitra
    Sustained-release ocular drug delivery systems : bench to bedside development / Susan S. Lee, Michael R. Robinson, and Scott M. Whitcup
    Ophthalmic examination as it pertains to general ocular toxicology : basic and advanced techniques and species-associated findings / David A. Wilkie
    Study design and methodologies for evaluation of anti-glaucoma drugs / Paul E. Miller
    Study design and methodologies for study of ocular medical devices / Joseph W. Carraway and Elaine M. Daniel
    Methodologies for microscopic characterization of ocular toxicity / Leandro B.C. Teixeira and James A. Render
    Nanoparticles for drug and gene delivery in treating diseases of the eye / Shreya S. Kulkarni and Uday B. Kompella.
    Digital Access Springer 2014
  • Article
    Tyras DH, Ahmad N, Kaiser GC, Barner HB, Codd JE, Willman VL.
    Ann Thorac Surg. 1979 Jun;27(6):547-53.
    Of 531 patients who underwent coronary artery bypass grafting during 1970 to 1973, 181 were restudied by ventriculography and by graft and coronary angiography at least 5 years following operation. Five patterns of postoperative ventricular function were identified: improved ventricular function resulting in normal left ventricular (LV) function; normal ventricular function that was unchanged; abnormal ventricular function that improved but did not reach normal; abnormal ventricular function that remained unchanged; and deterioration of LV function. Patients who regained (40) and those who retained normal ventricular function (49) comprise 49% of the series and patients with deterioration of ventricular function, only 20%. Graft patency and angina relief were significantly better in those with normal LV function than in those with LV deterioration. Progression of disease in grafted coronary arteries was similar in all groups, but was significantly higher in ungrafted coronary arteries (61.3%) in the patients showing deterioration than in either the improved patients or those with an unchanged normal LV (33.3% each) (p less than 0.05). The high incidence of progression of disease in ungrafted coronary arteries in the group with deterioration suggests that low graft patency and deterioration of ventricular function in this group might both be related to intrinsic acceleration of coronary atherosclerosis unrelated to operative intervention.
    Digital Access Access Options