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  • Book
    Giuseppe Lanzino, Robert F. Spetzler.
    Summary: A comprehensive overview of the basic science and current clinical knowledge on cavernous malformations of the brain and spinal cord. Cavernous Malformations of the Brain and Spinal Cord begins by covering general aspects of the disease, including the natural history, molecular biology, pathological processes, genetic basis, neuroradiology, and classification of cavernous malformations. Separate chapters then address the various types of cavernous malformations, thoroughly describing the clinical presentation and management approaches for each clinical situation. Incisive review of recent literature sheds light on topics of incidence, location, and outcomes. Neurosurgeons, neurologists, and neuroradiologists, especially those caring for patients with seizures and hemorrhagic stroke, will benefit from the information in this complete reference.

    Contents:
    Natural history of cavernous malformations of the central nervous system / Iman Feiz-Erfan ... [et al.]
    Molecular biology of cerebral cavernous malformations / Pascal Jabbour ... [et al.]
    Pathology of celebral malformations / Huan Wang and Meena Gujrati
    Genetics of cerebral cavernous malformations / E.W. Johnson and Joseph M. Zabramski
    Cavernous malformations as dynamic lesions: de novo formation, radiologic changes, and radiation-induced forms / Eugenio Pozzati
    Systemic cavernous malformations / Eugenio Pozzati
    Capillary telangiectasias, cavernous malformations, and developmental venous anomalies: different expressions of the same disease process? / Quoc-Anh Thai, Gustavo Pradilla, and Daniele Rigamonti
    Neuroradiology and classification of cavernous malformations / Lawrence C. Wang ... [et al.]
    Supratentorial cavernous malformations / Carlo Bortolotti ... [et al.]
    Deep-seated cerebral cavernous malformations / Uygur Er ... [et al.]
    Brain-stem cavernous malformations / Jeffrey D. Klopfenstein, Iman Feiz-Erfan, and Robert F. Spetzler
    Cavernous malformations of the spinal cord / Paolo Perrini ... [et al.]
    Giant cavernous angioma of the cavernous sinus / Atul Goel and Trimurti D. Nadkarni
    Management of atypical cavernous malformations / Vivek R. Deshmukh and Robert F. Spetzler
    Cavernous malformations and seizures: lesionectomy or epilepsy surgery? / Giovanni Broggi, Paolo Ferroli, and Angelo Franzini
    Cavernous malformations in children and adolescents / Carmine Mottolese ... [et al.]
    Radiosurgery for cavernous malformations / Douglas Kondziolka, John C. Flickinger, and L. Dade Lunsford.
    Digital Access Thieme MedOne Neurosurgery 2008
  • Article
    Byram JE, Doenhoff MJ, Musallam R, Brink LH, von Lichtenberg F.
    Am J Trop Med Hyg. 1979 Mar;28(2):274-85.
    Liver changes occurring in mice deprived of their T-cells by a combination of thymectomy and anti-mouse thymocyte serum, and in immunologically intact control mice, were followed during the early stages of heavy Schistosoma mansoni infections. Lesions in both groups began developing by day 38 and were maximal by day 48. Hepatic changes in control mice culminated in large hypersensitivity granulomas, tissue eosinophilia, portal periphlebitis, fibrosis, vascular obstruction, and infarction leading to arterialization and preferential sinusoidal channeling. Deprived mice showed greatly reduced egg reactions composed principally of macrophages, monocytes, and occasional neutrophils, and only minimal alteration of liver architecture; however, focal and disseminated hepatocellular lesions became prominent as the infections progressed, and by day 48 virtually every hepatocyte was affected. Typically, hepatocytes showed microvesicular cytoplasmic damage (steatosis) or ballooning degeneration with accompanying nuclear pyknosis or karyorrhexis. This cellular pathology may be attributed to the direct or indirect effect of eggs or egg products on liver cells. The administration of chronic infection serum obtained from immunocompetent mice to T-cell deprived mice dramatically eliminated the hepatocellular lesions. It also increased eosinophil participation and fibrosis in the egg reactions but did not restore the size and other cellular features typical of egg hypersensitivity granulomas. Serum from uninfected normal mice was found to lack these effects.
    Digital Access Access Options