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  • Book
    D'Agati, Vivette D.; Heptinstall, Robert H.; Jennette, J. Charles; Olson, Jean L.; Silva, Fred G.
    Contents:
    Renal anatomy and histology / Stephen M. Bonsib
    Development of the kidney / Adrian S. Woolf and Dagan Jenkins
    Primer on the pathologic classification and diagnosis of renal disease / J. Charles Jennette, Fred G. Silva, Jean L. Olson, and Vivette D. Dagati
    Cystic diseases and developmental kidney defects / Helen Liapis and Paul Winyard
    The nephrotic syndrome and minimal change disease / Jean L. Olson
    Focal segmental glomerulosclerosis and childhood nephrotic syndrome / Vivette D. DAgati and M. Barry Stokes
    Membranous glomerulonephritis / Glen S. Markowitz and Vivette D. DAgati
    Membranoproliferative glomerulonephritis / Xin J. Zhou and Fred G. Silva
    C3 glomerulopathies, including dense deposit disease / H. Terence Cook and Matthew C. Pickering
    Acute postinfectious glomerulonephritis and glomerulonephritis caused by persistent bacterial infection / Anjali A. Satoskar, Tibor Nadasdy, and Fred G. Silva
    Renal injury associated with human immunodeficiency virus infection and therapy / Arthur H. Cohen and Cynthia C. Nast
    Iga nephropathy and iga vasculitis (henoch-schonlein purpura) nephritis / Mark Haas
    Alports syndrome, familial benign hematuria, nail-patella syndrome, type iii collagen glomerulopathy, and piersons syndrome / Marie-Claire Gubler, Laurence Heidet, and Corinne Antignac
    Renal disease in systemic lupus erythematosus, mixed connective tissue disease, sjögren's syndrome, and rheumatoid arthritis / Vivette D. D'Agati and M. Barry Stokes
    Anti-glomerular basement membrane glomerulonephritis and goodpasture's syndrome / J. Charles Jennette and Volker Nickeleit
    Pauci-immune and antineutrophil cytoplasmic autoantibodymediated crescentic glomerulonephritis and vasculitis / J. Charles Jennette and David B. Thomas
    Volume 2
    Renal involvement in polyarteritis nodosa, kawasaki disease, takayasu arteritis, and giant cell arteritis / J. Charles Jennette and Harsharan K. Singh
    Thrombotic microangiopathies / Zoltan G. Laszik, Neeraja Kambham, and Fred G. Silva
    Renal disease in pregnancy / Agnes B. Fogo
    Renal disease caused by hypertension / Jean L. Olson
    Diabetic nephropathy / Jean L. Olson and Zoltan G. Laszik
    Renal diseases associated with plasma cell dyscrasias, amyloidoses, and waldenström's macroglobulinemia / Guillermo A. Herrera and Maria M. Picken
    Glomerular diseases with organized deposits / Samy S. Iskandar and Guillermo A. Herrera
    Pyelonephritis, reflux nephropathy, hydronephrosis, and nephrolithiasis / Helen Liapis, Joseph P. Gaut, John E. Tomaszewski, and Lois Arend
    Acute and chronic tubulointerstitial nephritis / Sergey V. Brodsky and Tibor Nadasdy
    Ischemic and toxic acute tubular injury and other ischemic renal injury / Gilbert Moeckel, Michael Kashgarian, and Lorraine V. Racusen
    Renal disease caused by inborn errors of metabolism, storage diseases, and hemoglobinopathies / Laura S. Finn
    Renal changes with aging and end-stage renal disease / Xin J. Zhou, Andrew Fenves, Nosratola D. Vaziri, and Ramesh Saxena
    Renal transplant pathology / Volker Nickeleit, Michael Mengel, and Robert B. Colvin
    Renal neoplasms / John N. Eble and David J. Grignon.
    Digital Access Ovid 2015
  • Article
    Logue GL, Silberman HR.
    Am J Med. 1979 Apr;66(4):703-6.
    A woman with chronic rheumatoid arthritis and severe granulocytopenia but without splenic enlargement by physical examination or radionuclide scanning was studied for granulocyte-bound immunoglobulin G (IgG) and serum antigranulocyte antibodies. Prior to splenectomy 73 to 110 X 10(-14) g/cell of IgG were detected on the patient's granulocytes, a value in the range (20 to 220 X 10(-14) g) found in 16 patients with classic Felty's syndrome. Granulocyte-bound IgG in 21 patients with rheumatoid arthritis without Felty's syndrome was less than 20 X 10(-14) g. Following splenectomy, the patient had a partial correction of her peripheral granulocyte count, and granulocyte bound IgG was repeated less than 20 X 10(-14) g/cell. When paraformaldehyde-fixed granulocytes, obtained either from normal donors or from the patient after splenectomy, were incubated in the patient's serum obtained before splenectomy, more IgG was bound than with control serums from patients with rheumatoid arthritis. Similar results were obtained when serums from patients with classic Felty's syndrome were incubated with paraformaldehyde-fixed granulocytes. Thus, patients with rheumatoid arthritis without overt splenic enlargement may have pathophysiologic Felty's syndrome, and in vitro studies such as these may be used to define this process.
    Digital Access Access Options