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  • Book
    Ralph Pantophlet, editor.
    Summary: Glycosylation is a common and extremely important modification in biological molecules, particularly of proteins. HIV Glycans in Infection and Immunity provides an overview of the roles of glycans in the transmission/infection, antigenicity, and immunogenicity of HIV and the HIV envelope glycoprotein. It explores recent advances in the understanding of the impact of HIV glycans in infection and their promise for immunological and therapeutic intervention. Novel collaborations between glycobiologists and immunologists in recent years have led to key advances in the understanding of HIV glycans. These cross-disciplinary endeavors, their achievements and their impact on the field are all addressed, herein.-- Source other than Library of Congress.

    Contents:
    HIV glycomics and glycoproteomics / Camille Bonomelli ... [et al.]
    Innate recognition of HIV-1 glycans : implications for infection, transmission, and immunity / Angelic M.G. van der Aar, Sonja I. Gringhuis, and teunis B.H. Geijtenbeek
    The influence of HIV enveloppe glycosylation on adaptive immune response / Catarina E. Hioe, Rajnish Kumar, and Shiu-Lok Hu
    Role of HIV glycans in transmission and immune escape / Penny L. Moore, Megan K. Murphy, and Cynthia A. Derdeyn
    Molecular recognition of HIV glycans by antibodies / Leopold Kong, Robyn L. Stanfield, and Ian A. Wilson
    Anti-carbohydrate HIV vaccine design / Lai-Xi Wang ... [et al.]
    Lectins as HIV microbicides / Leonardus M.I. Koharudin and Angela M. Gronenborn.
    Digital Access Springer 2014
  • Article
    Mitchell CJ, Humphrey CS, Bullen AW, Kelleher J.
    Scand J Gastroenterol. 1979;14(2):183-7.
    An oral pancreatic function test (PFT) using the synthetic peptide N-benzoyl-L-tyrosyl-p-aminobenzoic acid can assess pancreatic exocrine function, since urinary recovery of the ingested dose is an indirect index of chymotryptic activity. We have studied 34 subjects using this oral PFT, which correctly distinguished the control group (8 subjects) from the pancreatitis group (10 patients), results correlating well with Lundh test findings. However, the test was falsely abnormal on 9 out of 16 occasions in patients with bowel or liver disease. We therefore conclude that the present test cannot distinguish small-bowel disease from pancreatic disease, which is often the diagnostic problem, and is also frequently falsely abnormal in the presence of chronic liver disease.
    Digital Access Access Options