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  • Book
    Byung Ihn Choi, editor.
    Contents:
    Part 1. Pharynx/Esophagus
    1. Benign structural and functional abnormality of the esophagus, postop. Complication of the esophagus
    2. Esophagitis
    3. Benign and malignant tumor of the esophagus
    Part 2. Stomach/Duodenum
    4. Benign structural & functional abnormality of the stomach and duodenum
    5. Inflammatory and infectious diseases of the stomach and duodenum
    6. Mucosal tumor of the stomach
    7. Subepithelial tumor of the stomach
    8. Lymphoma and metastasis of the stomach
    9. Postoperative change and complications of the stomach and duodenum
    Part 3. Small Bowel
    10. Benign structural & functional abnormality of the small bowel
    11. Inflammatory disease of the small bowel
    12. Vasculitis and ischemic bowel disease
    13. Small bowel obstruction
    14. Benign tumor of the small bowel
    15. Malignant tumor of the small bowel
    Part 4. Colon
    16. Benign structural & functional abnormality of the colon
    17. Inflammatory & infectious disease of the colon
    18. Benign tumor of the colon
    19. Malignant tumor of the colon
    20. Postoperative change and complications of the colon
    Part 5. Mesentery, Peritoneum, abdominal wall
    21. Benign diseases of mesentery and omentum
    22. Malignant diseases of mesentery and omentum
    23. Abdominal wall abnormality & hernia.
    Digital Access Springer 2015
  • Article
    Winek CL, Marks MJ, Shanor SP, Davis ER.
    Clin Toxicol. 1978;13(2):281-96.
    The acute oral LD50 in Sprague-Dawley rats was determined to be 171 mg/kg (100-295) for males and 268 mg/kg (205-344) for females. 2. A 1 ppm dietary supplement of Vancide KS for 90 days did not induce any abnormalities in weanling Sprague-Dawley rats. The parameters evaluated were serum glutamic-oxaloacetic transaminase (SGOT), hematocrit, differential white blood cell count, food consumption, and weight gain, along with histologic studies of the myocardium, spleen, liver, kidney, stomach, and small intestine. A 500 ppm diet was lethal. Weanling and older rats subjected to 1000 and 10,000 ppm diets died within 5 days. 3. Vancide KS induced no acute dermal toxicity, nor did it exhibit percutaneous absorption in New Zealand strain albino rabbits. 4. Vancide KS induced no chronic dermal toxicity in New Zealand strain albino rabbits. 5. Vancide KS was not shown to be teratogenic. It exhibited an antifertility action, especially in Sprague-Dawley rats dosed on day 1 through day 7 of timed-pregnancy. 6. Vancide KS was shown to be an eye irritant which induces corneal opacity. 7. Acute oral toxicity studies in Sprague-Dawley rats indicate that Vancide KS should be classified as a toxic substance as defined in the regulations under the Federal Hazardous Labeling Act. 8. The intravenous administration of 25 mg/kg of Vancide KS to New Zealand strain albino rabbits induced death preceded by topic convulsions. 9. Vancide KS did not induce skin sensitization in male adult guinea pigs.
    Digital Access Access Options