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- BookLori A. Erickson.Summary: "Atlas of Endocrine Pathology provides a comprehensive compendium of photomicrographs of common and uncommon entities in endocrine pathology. The volume includes histologic features of normal features, reactive conditions, hyperplasia, and tumors. The most helpful diagnostic features are illustrated to provide direction and clues to the diagnosis of endocrine tumors. Furthermore, photomicrographs highlight the most pertinent diagnostic features in problematic diagnoses in endocrine pathology. Authored by a nationally and internationally recognized pathologist, Atlas of Endocrine Pathology is an important learning tool for those becoming familiar with the diverse entities encountered in endocrine pathology and a valuable reference for practicing pathologists faced with challenging diagnoses in endocrine pathology."--Publisher's website.
Contents:
1. Thyroid Histology
2. Thyroiditis
3. Graves Disease (Diffuse Hyperplasia)
4. Thyroid Goiter
5. Papillary Thyroid Carcinoma
6. Hyalinizing Trabecular Tumor
7. Follicular Thyroid Neoplasms
8. Hurthle Cell Thyroid Neoplasms
9. Poorly Differentiated Thyroid Carcinoma
10. Anaplastic Thyroid Carcinoma
11. Tumors of C Cells
12. Unusual Thyroid Tumors
13. Metastases to Thyroid
14. Parathyroid Histology
15. Parathyroid Hyperplasia
16. Parathyroid Adenoma
17. Parathyroid Carcinoma
18. Parathyromatosis
19. Metastases to Parathyroid
20. Adrenal Gland Histology
21. Adrenal Cysts
22. Adrenal Cortical Hyperplasia
23. Adrenal Cortical Adenoma
24. Adrenal Cortical Carcinoma
25. Adrenal Myelolipoma
26. Pheochromocytoma and Adrenal Medullary Hyperplasia
27. Ganglioneuroma and Neuroblastoma
28. Unusual Adrenal Tumors
29. Metastases to the Adrenal GlandDigital Access Springer 2014 - ArticleMarbrook J, Nawa Y, Miller JF.J Exp Med. 1978 Jul 01;148(1):324-8.By using a culture system that allows the segregation of individual precursors of cytotoxic lymphocytes, the number of clones generated by cells from different combinations of congenic mice have been measured. It has been found that 0.3% of the total anti-H2d clones are generated by stimulators which differ predominantly at the H-2 locus. The contribution of non-H-2 antigens to anti-H-2 responses is discussed.