Today's Hours: 12:00pm - 8:00pm

Search

Did You Mean:

Search Results

  • Book
    edited by Jon Lorsch.
    Contents:
    Computational prediction of RNA secondary structure
    Reverse-transcription PCR (RT-PCR)
    Northern blotting
    Explanatory chapter: nuclease protection assays
    In vitro transcription from plasmid or PCR-amplified DNA
    Measurement of in vivo synthesis rates
    Method for measuring mRNA decay rate in saccharomyces cerevisiae
    Analysis of polysomes from bacteria
    Polysome profile analysis - yeast
    Polysome analysis of mammalian cells
    Polysome analysis for determining mRNA and ribosome association in saccharomyces cerevisiae
    Oligo(dT)-primed RT-PCR isolation of polyadenylated RNA degradation intermediates
    Circularized RT-PCR (cRT-PCR): analysis of the 5' ends, 3' ends, and poly(A) tails of RNA
    RNA radiolabeling
    Fluorescently labeling synthetic RNAs
    Analysis of RNA by analytical polyacrylamide gel
    RNA purification by preparative polyacrylamide gel
    Explanatory chapter: nucleic acid concentration determination
    RNA purification-precipitation methods
    Reverse transcriptase dideoxy sequencing of RNA
    RNA structure experimental analysis-chemical modification
    Structural analysis of RNA backbone using in-line probing.
    Digital Access ScienceDirect 2013
  • Article
    Nagaoka K, Nabeya N, Sakurami T, Imura H, Kuno S.
    Nihon Naibunpi Gakkai Zasshi. 1978 Jan 20;54(1):36-42.
    There is an increasing evidence that autoimmune mechanisms may have a role in the pathogenesis in insulin-dependent diabetics. The numerical and functional study of peripheral blood lymphocytes in diabetes mellitus might indirectly contribute to the understanding of its pathogenesis. In this study, detection of peripheral blood T lymphocytes was measured by rosettes with sheep red blood cells (SRBC), and B lymphocytes were measured by immunofluorescence with specific antiserum to immunoglobulins. The mean (+/- SD) percentage of SRBC was 67.6 +/- 7.2 in 21 normal subjects, 71.5 +/- 7.0 in 15 insulin-dependent diabetics, and 68.6 +/- 6.7 in 30 insulin-independent diabetics. There was no difference in the absolute T-lymphocyte number per mm3 in these three groups. Insulin-dependent diabetics showed a normal percentage and absolute number of B lymphocytes when compared with normal subjects.
    Digital Access Access Options