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  • Book
    Benhur Lee, Paul A. Rota, editors.
    Contents:
    Introduction: Nipah Virus--Discovery and Origin / Kaw Bing Chua
    Ecological Aspects of Hendra Virus / Hume Field, Gary Crameri, Nina Yu-Hsin Kung and Lin-Fa Wang
    Epidemiology of Henipavirus Disease in Humans / Stephen P. Luby and Emily S. Gurley
    Molecular Virology of the Henipaviruses / Paul A. Rota and Michael K. Lo
    Henipavirus Receptor Usage and Tropism / Olivier Pernet, Yao E Wang and Benhur Lee
    Henipavirus Membrane Fusion and Viral Entry / Hector C. Aguilar and Ronald M. Iorio
    Clinical and Pathological Manifestations of Human Henipavirus Infection / K. T. Wong and C. T. Tan
    Henipaviruses in Their Natural Animal Hosts / D. J. Middleton and H. M. Weingartl
    Nipah and Hendra Virus Interactions with the Innate Immune System / Christopher F. Basler
    Animal Challenge Models of Henipavirus Infection and Pathogenesis / Thomas W. Geisbert, Heinz Feldmann and Christopher C. Broder
    Diagnosis of Henipavirus Infection: Current Capabilities and Future Directions / Lin-Fa Wang and Peter Daniels
    Immunization Strategies Against Henipaviruses / Christopher C. Broder, Thomas W. Geisbert, Kai Xu, Dimitar B. Nikolov and Lin-Fa Wang, et al.
    Digital Access Springer 2012
    Access via Current topics in microbiology and immunology ; 2012; 359
    Location
    Version
    Call Number
    Items
    Current topics in microbiology and immunology ; 2012; 359
    2012
  • Article
    Liakos GM, Casey TA.
    Br J Ophthalmol. 1978 Jan;62(1):39-45.
    Seven cases with posterior polymorphous changes of the cornea are reported. After clinical and pathological examination of the above cases, as well as a short review of the literature, the following points are made: (1) Some cases are congenital, being either familial or sporadic, but others are acquired. (2) The term "posterior polymorphous keratopathy" covers all the variants of the condition and is preferred to the traditional "posterior polymorphous dystrophy". (3) The congenital type is a mild variant of the mesodermal dysplasia, whereas the acquired type follows local disease. (4) The condition can be static, but over 50% of cases are slowly progressive, calling for penetrating keratoplasty.
    Digital Access Access Options