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- BookArmand B. Cognetta Jr., William M. Mendenhall, editors.Contents:
History of radiation therapy in dermatology
Radiobiology
Physical aspects of dermatological radiotherapy
Radiation protection
Staging of squamous cell carcinoma and basal cell carcinoma
Acute and chronic cutaneous reactions to radiotherapy
Efficacy of superficial radiotherapy
Treatment selection for superficial radiotherapy
Superficial radiation therapy treatment planning
Current use of dermatologic radiotherapy in the United States
Grenz ray therapy
Radiotherapy for cutaneous squamous and basal cell carcinomas
Brachytherapy
Cutaneous merkel cell carcinoma
Radiotherapy for cutaneous angiosarcoma
Radiotherapy for dermatofibrosarcoma protuberans
Squamous and basal cell carcinomas with perineural invasion
Getting started in superficial radiation for the dermatology practice.Digital Access Springer 2013 - ArticleCouch JR, Abdou NI, Sagawa A.Neurology. 1978 Feb;28(2):119-23.Histoplasma meningitis usually occurs in conjunction with disseminated histoplasmosis. We studied a patient with common variable hypogammaglobulinemia who manifested meningitis without disseminated histoplasmosis. No histoplasma antibody was detected in cerebrospinal fluid (CSF) or blood. Evaluation of lymphocyte function in the blood revealed normal numbers of T cells with increased numbers of B cells. Most blood lymphocytes were identifiable, but most lymphocytes in CSF were null cells. Lymphocyte proliferative response to phytohemagglutinin or pokeweed mitogen was poor. T cells in CSF suppressed proliferative responses to histoplasma antigen by cells from blood or CSF, whereas T cells from blood did not. This difference suggested compartmentalization of T-cell function. The lack of humoral and cellular response to histoplasma in CSF may have allowed meningitis to develop, while the cellular response to histoplasma elsewhere prevented development of disseminated histoplasmosis.