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  • Book
    volume editors, Hiromichi Suzuki, Hiroyuki Hirasawa.
    Summary: "Describes the use of hemodiafiltration to inhibit the cytokine storms which cause serious organ damage in patients with septic shock. Moreover, the technical construction of the blood purification system, which includes various machines, devices, membranes, fluids, etc., is explained in detail. Finally, leading experts discuss the concept of continuous renal replacement therapy as the standard care in critically ill patients with severe acute kidney injury."--Website.

    Contents:
    Current status of blood purification in critical care in Japan / Kaizu, K. ... [et al.]
    Terminology and classification of blood purification in critical care in Japan / Kawanishi, H.
    Indications for blood purification in critical care / Hirasawa, H.
    Acute kidney injury of non-septic origin requiring dialysis therapy / Suzuki, H. ... [et al.]
    Septic acute renal failure / Mori, T.; Shimizu, T.; Tani, T.
    Non-renal indications for continuous renal replacement therapy : current status in Japan / Oda, S. ... [et al.]
    Continuous hemodiafiltration using a polymethyl methacrylate membrane hemofilter for severe acute pancreatitis / Abe, R. ... [et al.]
    Blood purification in fulminant hepatic failure / Shinozaki, K. ... [et al.]
    Treatment of severe sepsis and septic shock by CHDF using a PMMA membrane hemofilter as a cytokine modulator / Nakamura, M. ... [et al.]
    Efficacy of continuous hemodiafiltration with a cytokine-adsorbing hemofilter in the treatment of acute respiratory distress syndrome / Matsuda, K. ... [et al.]
    Blood purification for intoxication / Nakae, H. Current progress in blood purification methods used in critical care Medicine / Saito, A.
    Membrane materials for blood purification in critical care / Yamashita, A.C.; Tomisawa, N.
    Anticoagulation in acute blood purification for acute renal failure in critical care / Shinoda, T.
    Equipment and monitoring in continuous renal replacement therapy / Yamashita, Y. ... [et al.]
    Cytokine adsorbing columns / Taniguchi, T.
    Plasma dia-filtration for severe sepsis / Eguchi, Y.
    Application of polymycin b convalently immobilized fiber in patients with septic shock / Suzuki, H.; Shoji, H.
    Special considerations in continuous hemodiafiltration with critically ill pediatric patients / Shiga, H. ... [et al.]
    Selection of modality in continuous renal replacement therapy / Kanno, Y.; Suzuki, H.
    Online CHDF system : excellent cost-effectiveness for continuous renal replacement therapy with high efficacy and individualization / Takatori, M. ... [et al.]
    'Super high-flux' or 'high cut-off' hemofiltration and hemodialysis / Naka, T.; Haase, M.; Bellomo, R.
    Digital Access Karger 2010
  • Article
    Barbieri RL, Canick JA, Ryan KJ.
    Endocrinology. 1977 Dec;101(6):1676-82.
    The effects of danazol on steroidogenesis in vitro in the rat testis were examined by studying: 1) androgen synthesis in rat Leydig cells cultured with danazol, 2) danazol binding to rat testis microsomal cytochrome P-450, and 3) enzyme kinetics of danazol inhibition of the microsomal enzymes of testicular steroidogenesis. Concentrations of danazol as low as 1 micrometer suppressed LH-stimulated testosterone and androstenedione production in cultured Leydig cells. The addition of danazol to a preparation of testicular microsomes elicited a type I cytochrome P-450 binding spectrum, with an apparent spectral dissociation constant (Ks) of 4.8 micrometer. Danazol inhibited progesterone and 17alpha-hydroxy-progesterone binding to microsomal P-450 with apparent spectral inhibition constants of 2.4 micrometer and 2.8 micrometer, respectively. Danazol competitively inhibited 3beta-hydroxy-delta5-steroid dehydrogenase-isomerase (apparent enzymatic inhibition constant, KI = 5.8 micrometer), 17alpha-hydroxylase (KI = 2.4 micrometer), 17,20 lyase (KI = 1.9 micrometer), and 17beta-hydroxysteroid dehydrogenase (KI = 4.4 micrometer). These findings indicate that low concentrations of danazol directly inhibit steroidogenesis in the rat testis in vitro.
    Digital Access Access Options