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  • Book
    Yu-Kang Tu, Darren C. Greenwood, editors.
    Contents:
    Confounding and causal path diagrams
    Statistical modelling of partially observed data using multiple imputation: principles and practice
    Measurement errors in epidemiology
    Selection bias in epidemiologic studies
    Multilevel modelling
    Modelling data that exhibit an excess number of zeros : zero-inflated models and generic mixture models
    Multilevel latent class modelling
    Bayesian bivariate disease mapping
    Multivariate random fraility effects model for multiple spatially dependent survival data
    Meta-analysis of observational studies
    Directed acyclic graphs and structural equation modelling
    Latent growth curve models
    Growth mixture modelling for life course epidemiology
    G-estimation for accelerated failure time models
    Generalised additive models
    Regression and classification trees
    Statistical interactions and gene-environment joint effects.
    Digital Access Springer 2012
  • Article
    Wolberg WH.
    Cancer Res. 1977 Oct;37(10):3711-9.
    Colonic tissue membrane binding to peripheral blood mononuclear leukocytes was quantitated by 125I labeling of membrane fragments and by determining the acquisition of membrane-specific enzyme activity and radioactivity in mononuclear cells after contact with the tissue membrane fragments. Mononuclear cells bound equal amounts of normal and tumor tissue membrane fragments. Mononuclear cells capable of binding homologous but not autologous colonic tissue membranes were recovered from the peripheral blood of colon cancer-bearing patients. Mononuclear cells capable of binding autologous colonic tissue membranes appeared in the peripheral blood of patients after curative but not palliative tumor resection. Tumor membrane enzymes, including alkaline phosphatase, were introduced to mononuclear cells by bound tissue fragments. The activity of alkaline phosphatase present in the bound membrane fragments was inhibited by the immunorestorative drug, levamisole. Cellular debris liberated from tumors may play an important role in overcoming the host's defenses by binding to mononuclear cells, saturating antigen-binding sites, and introducing exogenous enzymes.
    Digital Access Access Options